Tomohito Doke

Assistant Professor

Nagoya University Graduate School of Medicine

Japan

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Tomohito Doke is an Assistant Professor at Nagoya University Graduate School of Medicine in Japan. His research focuses on kidney disease, with recent publications exploring topics such as lipid metabolism, ferroptosis, and the mechanisms behind diabetic kidney disease. Through single-cell transcriptomics and proteomics, he aims to identify biomarkers and therapeutic targets for improving kidney health.

Articles (15)

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2025

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2021

Collaborators (6)

Hongbo Liu

Assistant Professor

University of Rochester

UNITED STATES

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Daniel Aldridge

Columbia University

UNITED STATES

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Lauren Elizabeth Lee

University of Pennsylvania

UNITED STATES

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Benjamin F. Voight

UNITED STATES

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Katalin Susztak

Professor

University of Pennsylvania

UNITED STATES

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Joseph Baur

Associate Professor

University of Pennsylvania

UNITED STATES

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Tomohito Doke

Research Interests

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Articles (15)

Glutathione-specific gamma–glutamylcyclotransferase 1 ( CHAC1 ) increases kidney disease risk by modulating ferroptosis

The key role of altered tubule cell lipid metabolism in kidney disease development

ACSS2 gene variants determine kidney disease risk by controlling de novo lipogenesis in kidney tubules

Endothelial–Mesenchymal Transition Switch and Diabetic Kidney Disease

Single-cell transcriptomics and chromatin accessibility profiling elucidate the kidney-protective mechanism of mineralocorticoid receptor antagonists

Unbiased Human Kidney Tissue Proteomics Identifies Matrix Metalloproteinase 7 as a Kidney Disease Biomarker

NAD+ precursor supplementation prevents mtRNA/RIG-I-dependent inflammation during kidney injury

Molecular pathways that drive diabetic kidney disease

Single-cell analysis highlights differences in druggable pathways underlying adaptive or fibrotic kidney regeneration

Epigenomic and transcriptomic analyses define core cell types, genes and targetable mechanisms for kidney disease

Single-cell analysis identifies the interaction of altered renal tubules with basophils orchestrating kidney fibrosis

Genome-wide association studies identify the role of caspase-9 in kidney disease

NAD+ flux is maintained in aged mice despite lower tissue concentrations.

Transcriptome-wide association analysis identifies DACH1 as a kidney disease risk gene that contributes to fibrosis

DACH1 protects podocytes from experimental diabetic injury and modulates PTIP-H3K4Me3 activity.

Collaborators (6)

Hongbo Liu

Daniel Aldridge

Lauren Elizabeth Lee

Benjamin F. Voight

Katalin Susztak

Joseph Baur

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