Toshinori Hyodo

Senior Assistant Professor

Aichi Medical University

Japan

Research Interests

Explore related searches

Professors in Japan Professors in Immunology Immunology professors in Japan

Contact this professor

Get a free first email built on one of their papers, ready for you to edit and send yourself.

About

Toshinori Hyodo is a Senior Assistant Professor at Aichi Medical University in Japan. His research primarily focuses on genome editing technologies, cellular dynamics in cancer, and the molecular mechanisms of stress granule formation. Recent publications include studies on CRISPR/Cas9-mediated targeted knock-in, the role of ARK5 in multiple myeloma cells, and novel molecular targets for malignant mesothelioma.

Articles (17)

Year:

2025

Year:

2024

Year:

2023

Year:

2023

Year:

2023

Year:

2023

Year:

2023

Year:

2022

Year:

2022

Year:

2021

Year:

2021

Year:

2021

Year:

2021

Year:

2020

Year:

2020

Year:

Aichi Medical University

JAPAN

View details

ApplyKite TwinLearns as you apply

Make your next move.

Prepare your application

Review drafts before you send them.

Toshinori Hyodo

Research Interests

Explore related searches

Contact this professor

About

Articles (17)

Characterization of targeted knock-in achieved via tandem paired nicking mediated by CRISPR/Cas9 nickases.

ARK5 enhances cell survival associated with mitochondrial morphological dynamics from fusion to fission in human multiple myeloma cells

CAMK2D: a novel molecular target for BAP1-deficient malignant mesothelioma

PDZ‐binding kinase inhibitor OTS514 suppresses the proliferation of oral squamous carcinoma cells

Leucine zipper protein 1 (LUZP1) regulates the constriction velocity of the contractile ring during cytokinesis

Flow cytometry-based quantification of genome editing efficiency in human cell lines using the L1CAM gene.

The association of UBAP2L and G3BP1 mediated by small nucleolar RNA is essential for stress granule formation.

BCL6 inhibition ameliorates ruxolitinib resistance in CRLF2-rearranged acute lymphoblastic leukemia.

Correction of a CD55 mutation to quantify the efficiency of targeted knock-in via flow cytometry.

Flow cytometry-based quantification of targeted knock-in events in human cell lines using a GPI-anchor biosynthesis gene PIGP.

Experimental strategies to achieve efficient targeted knock-in via tandem paired nicking.

CD52 is a novel target for the treatment of FLT3-ITD-mutated myeloid leukemia

PBK Enhances Cellular Proliferation With Histone H3 Phosphorylation and Suppresses Migration and Invasion With CDH1 Stabilization in Colorectal Cancer.

Biallelic loss of FAM46C triggers tumor growth with concomitant activation of Akt signaling in multiple myeloma cells

Identification of CD24 as a potential diagnostic and therapeutic target for malignant pleural mesothelioma.

Novel Interleukin-6 Inducible Gene PDZ-Binding Kinase Promotes Tumor Growth of Multiple Myeloma Cells.

Targeting MEF2D-fusion Oncogenic Transcriptional Circuitries in B-cell Precursor Acute Lymphoblastic Leukemia.

Collaborators (6)

Sivasundaram Karnan

Shinobu Tsuzuki

Shingo Inaguma

Akinobu Ota

Kaori Uchino

Hiroyuki Konishi

Make your next move.