Tsuneo Imanaka

Visiting Professor

Hiroshima University

Japan

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Tsuneo Imanaka is a Visiting Professor at Hiroshima International University in Japan. His research focuses on the roles of various genetic mutations and transport mechanisms in cellular metabolism and disease, particularly in relation to lipids and mitochondrial function. Recent publications include studies on the interaction of SBDS gene mutations with cellular processes, the mechanisms of cobalamin transport, and the characterization of cell lines relevant to X-linked adrenoleukodystrophy.

Articles (12)

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2024

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2024

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2023

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2023

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2023

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2022

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2022

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2021

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2021

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2021

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2021

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2020

Collaborators (4)

Shiro Watanabe

Associate Professor

University of Toyama

JAPAN

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Takanori So

Professor

JAPAN

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Atsushi Yamashita

Professor

Teikyo University

JAPAN

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Kosuke Kawaguchi

JAPAN

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Tsuneo Imanaka

Research Interests

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Articles (12)

FXR Antagonist FLG249 Lowers Hepatic Triacylglycerol and Serum Cholesterol Level in High-Fat Diet-Induced Obese Mice.

SBDS Gene Mutation Increases ROS Production and Causes DNA Damage as Well as Oxidation of Mitochondrial Membranes in the Murine Myeloid Cell Line 32Dcl3.

Behavior of intracellular lipid droplets during cell division in HuH7 hepatoma cells.

Transmembrane helix 6 of ABCD4 is indispensable for cobalamin transport.

M phase-specific interaction between SBDS and RNF2 at the mitotic spindles regulates mitotic progression.

SBDS interacts with RNF2 and is degraded through RNF2-dependent ubiquitination.

Substrate Specificity and the Direction of Transport in the ABC Transporters ABCD1-3 and ABCD4.

Generation of an immortalized astrocytic cell line from Abcd1-deficient H-2KbtsA58 mice to facilitate the study of the role of astrocytes in X-linked adrenoleukodystrophy

Acyl-CoA thioesterase activity of peroxisomal ABC protein ABCD1 is required for the transport of very long-chain acyl-CoA into peroxisomes.

Bone marrow transplantation into Abcd1-deficient mice: Distribution of donor derived-cells and biological characterization of the brain of the recipient mice.

The lysosomal protein ABCD4 can transport vitamin B12 across liposomal membranes in vitro

A novel dynein-type AAA+ protein with peroxisomal targeting signal type 2.

Collaborators (4)

Shiro Watanabe

Takanori So

Atsushi Yamashita

Kosuke Kawaguchi

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