Vienna Brunt

Assistant Professor

University of Colorado Anschutz Medical Campus

United States

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Vienna Brunt is an Assistant Professor at Anschutz Medical Campus, University of Colorado, USA. Her research focuses on cardiovascular health, cellular senescence, and the interplay between the gut microbiome and arterial function. Recent publications include investigations into the effects of doxorubicin chemotherapy on aortic stiffness and the impact of exogenous estradiol on cardiovagal baroreflex sensitivity. She is also exploring novel approaches to understanding arterial aging and dysfunction in various health contexts.

Articles (18)

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2025

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2025

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2025

Changes in the circulating milieu with advancing age may be a mechanism underlying age-related arterial dysfunction. Ex vivo exposure of young mouse arteries to the circulating milieu from old mice or midlife/older adults impairs arterial function whereas exposure of old mouse arteries to the circulating milieu from young mice or young adults improves arterial function. These findings establish that the circulating milieu directly influences arterial function with aging.

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2024

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2024

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2024

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2024

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2024

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2023

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2022

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2021

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2021

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2021

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2021

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2021

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2021

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2020

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2020

Collaborators (8)

Yara Bernaldo de Quirós

Universidad de Las Palmas de Gran Canaria

SPAIN

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Zachary S. Clayton

University of Colorado Boulder

UNITED STATES

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Thomas J. LaRocca

UNITED STATES

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Michael Widlansky

Professor and Director

Medical College of Wisconsin

UNITED STATES

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Marissa A. Ehringer

UNITED STATES

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Doug Seals

Distinguished Professor

University of Colorado Boulder

UNITED STATES

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Kanokwan Bunsawat

Research Assistant Professor

University of Utah

UNITED STATES

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John Halliwill

Assistant, Associate, and Full Professor

University of Oregon

UNITED STATES

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Vienna Brunt

Research Interests

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About

Articles (18)

Cellular Senescence Mediates Doxorubicin Chemotherapy-Induced Aortic Stiffening: Role of Glycation Stress

Exogenous estradiol increases cardiovagal baroreflex sensitivity during a hypertensive stimulus in premenopausal young women

Endothelial dysfunction in chronic kidney disease: a clinical perspective

Role of the circulating milieu in age-related arterial dysfunction: a novel ex vivo approach

The bottlenose dolphin (Tursiops truncatus): a novel model for studying healthy arterial aging

The gut microbiome as a modulator of arterial function and age-related arterial dysfunction

Untangling Complexity: Gut Microbiota, Metabolites, and Fiber Intake in Type 2 Diabetes

Intermittent supplementation with fisetin improves arterial function in old mice by decreasing cellular senescence

Circulating interleukin-37 declines with aging in healthy humans: relations to healthspan indicators and IL37 gene SNPs

Suppression of trimethylamine N-oxide with DMB mitigates vascular dysfunction, exercise intolerance, and frailty associated with a Western-style diet in mice

Brachial and carotid hemodynamic response to hot water immersion in men and women

Gut Microbiome-Derived Metabolite Trimethylamine N-Oxide Induces Aortic Stiffening and Increases Systolic Blood Pressure With Aging in Mice and Humans

Apigenin restores endothelial function by ameliorating oxidative stress, reverses aortic stiffening, and mitigates vascular inflammation with aging

Heat therapy: mechanistic underpinnings and applications to cardiovascular health

Tumor Necrosis Factor Alpha-Mediated Inflammation and Remodeling of the Extracellular Matrix Underlies Aortic Stiffening Induced by the Common Chemotherapeutic Agent Doxorubicin

The gut microbiome–derived metabolite trimethylamine N-oxide modulates neuroinflammation and cognitive function with aging

Attenuation of exaggerated blood pressure responses to exercise: a byproduct of improved vascular function with passive heat therapy?

Preventing endothelial cell‐mediated muscle satellite cell dysfunction: a new hot topic?

Collaborators (8)

Yara Bernaldo de Quirós

Zachary S. Clayton

Thomas J. LaRocca

Michael Widlansky

Marissa A. Ehringer

Doug Seals

Kanokwan Bunsawat

John Halliwill

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