Vigo Heissmeyer

Professor

Ludwig-Maximilians-Universität München

Germany

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About

Vigo Heissmeyer is a Professor at Ludwig-Maximilians-Universität München, Germany. His research primarily focuses on immunology, cellular signaling, and the mechanisms of autoimmunity. Recent publications include studies on ferroptosis, the regulation of T cell fate, and the interplay of various proteins in cytokine expression and stability.

Articles (17)

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2025

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2025

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2024

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2024

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2024

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2024

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2022

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2022

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2021

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2021

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2021

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2020

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2020

Collaborators (11)

Ken Bracke

Associate Professor

Ghent University

BELGIUM

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Silvia Monticelli

Lecturer

ETH Zürich

SWITZERLAND

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Michael Sattler

Technical University of Munich

GERMANY

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Tobias Straub

Head

Ludwig-Maximilians-Universität München

GERMANY

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Axel Kallies

University of Melbourne

AUSTRALIA

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Taku Kureha

Lecturer

University of Tokyo

JAPAN

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Dirk Baumjohann

Associate Professor

University of Bonn

GERMANY

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Lukas Jeker

Tenure Track Assistant Professor/Principal Investigator

University of Basel

SWITZERLAND

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Cristina Leoni

Institute for Research in Biomedicine

SWITZERLAND

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Naoto Kawakami

University Hospital

GERMANY

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Hyun-Seo Kang

Ludwig-Maximilians-Universität München

GERMANY

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Vigo Heissmeyer

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Articles (17)

MALT1 inhibitor MI-2 induces ferroptosis by direct targeting of GPX4.

Regulation and Function of the Atypical IκBs-Bcl-3, IκB<sub>NS</sub>, and IκBζ-in Lymphocytes and Autoimmunity.

The thymocyte-specific RNA-binding protein Arpp21 provides TCR repertoire diversity by binding to the 3’-UTR and promoting Rag1 mRNA expression

Correction to “The thymocyte-specific RNA-binding protein Arpp21 provides TCR repertoire diversity by binding to the 3’-UTR and promoting Rag1 mRNA expression”

TGF-β specifies TFH versus TH17 cell fates in murine CD4+ T cells through c-Maf.

Aloperine Suppresses Cancer Progression by Interacting with VPS4A to Inhibit Autophagosome-lysosome Fusion in NSCLC.

Crosstalk between Regnase-1 and -3 shapes mast cell survival and cytokine expression.

The mRNA methyltransferase Mettl3 modulates cytokine mRNA stability and limits functional responses in mast cells

Unrestrained cleavage of Roquin-1 by MALT1 induces spontaneous T cell activation and the development of autoimmunity.

Null and missense mutations of ERI1 cause a recessive phenotypic dichotomy in humans.

The arginine methyltransferase PRMT7 promotes extravasation of monocytes resulting in tissue injury in COPD.

Cooperation of RNA-Binding Proteins - a Focus on Roquin Function in T Cells.

Disrupting Roquin-1 interaction with Regnase-1 induces autoimmunity and enhances antitumor responses.

TRAF6 prevents fatal inflammation by homeostatic suppression of MALT1 protease.

Defining the RBPome of primary T helper cells to elucidate higher-order Roquin-mediated mRNA regulation.

Elevated Exhaustion Levels of NK and CD8<sup>+</sup> T Cells as Indicators for Progression and Prognosis of COVID-19 Disease.

Validation strategies for antibodies targeting modified ribonucleotides.

Collaborators (11)

Ken Bracke

Silvia Monticelli

Michael Sattler

Tobias Straub

Axel Kallies

Taku Kureha

Dirk Baumjohann

Lukas Jeker

Cristina Leoni

Naoto Kawakami

Hyun-Seo Kang

Make your next move.

Vigo Heissmeyer

Research Interests

Explore related searches

Contact this professor

About

Articles (17)

MALT1 inhibitor MI-2 induces ferroptosis by direct targeting of GPX4.

Regulation and Function of the Atypical IκBs-Bcl-3, IκB&lt;sub&gt;NS&lt;/sub&gt;, and IκBζ-in Lymphocytes and Autoimmunity.

The thymocyte-specific RNA-binding protein Arpp21 provides TCR repertoire diversity by binding to the 3’-UTR and promoting Rag1 mRNA expression

Correction to “The thymocyte-specific RNA-binding protein Arpp21 provides TCR repertoire diversity by binding to the 3’-UTR and promoting Rag1 mRNA expression”

TGF-β specifies TFH versus TH17 cell fates in murine CD4+ T cells through c-Maf.

Aloperine Suppresses Cancer Progression by Interacting with VPS4A to Inhibit Autophagosome-lysosome Fusion in NSCLC.

Crosstalk between Regnase-1 and -3 shapes mast cell survival and cytokine expression.

The mRNA methyltransferase Mettl3 modulates cytokine mRNA stability and limits functional responses in mast cells

Unrestrained cleavage of Roquin-1 by MALT1 induces spontaneous T cell activation and the development of autoimmunity.

Null and missense mutations of ERI1 cause a recessive phenotypic dichotomy in humans.

The arginine methyltransferase PRMT7 promotes extravasation of monocytes resulting in tissue injury in COPD.

Cooperation of RNA-Binding Proteins - a Focus on Roquin Function in T Cells.

Disrupting Roquin-1 interaction with Regnase-1 induces autoimmunity and enhances antitumor responses.

TRAF6 prevents fatal inflammation by homeostatic suppression of MALT1 protease.

Defining the RBPome of primary T helper cells to elucidate higher-order Roquin-mediated mRNA regulation.

Elevated Exhaustion Levels of NK and CD8<sup>+</sup> T Cells as Indicators for Progression and Prognosis of COVID-19 Disease.

Validation strategies for antibodies targeting modified ribonucleotides.

Collaborators (11)

Ken Bracke

Silvia Monticelli

Michael Sattler

Tobias Straub

Axel Kallies

Taku Kureha

Dirk Baumjohann

Lukas Jeker

Cristina Leoni

Naoto Kawakami

Hyun-Seo Kang

Make your next move.

Regulation and Function of the Atypical IκBs-Bcl-3, IκB<sub>NS</sub>, and IκBζ-in Lymphocytes and Autoimmunity.