Wayne Phillips

Adjunct Professor

Monash Health

Australia

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About

Wayne Phillips is an Adjunct Professor at Monash Health in Australia. His research focuses on cancer biology and metabolic processes, particularly related to the PI3K signaling pathway. Recent work includes studies on mutations in the Pik3ca gene, the development of mouse models for intestinal adenocarcinoma, and the role of immune cells in cancer progression. He has published extensively in areas such as ferroptosis and the interaction of dietary factors with cancer treatment.

Articles (19)

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2025

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2021

Collaborators (15)

Dmitri Mouradov

Walter and Eliza Hall Institute

AUSTRALIA

Wayne Phillips

Research Interests

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Articles (19)

Ubiquitous expression of an activating mutation in the Pik3ca gene reprograms glucose and lipid metabolism in mice

Caspase-2 protects against ferroptotic cell death.

Targeting a STING agonist to perivascular macrophages in prostate tumors delays resistance to androgen deprivation therapy.

Development of an in vivo syngeneic mouse transplant model of invasive intestinal adenocarcinoma driven by endogenous expression of Pik3caH1047R and Apc loss

Direct activation of PI3K in osteoblasts and osteocytes strengthens murine bone through sex-specific actions on cortical surfaces

YBX1 integration of oncogenic PI3K/mTOR signalling regulates the fitness of malignant epithelial cells

PLEKHS1 drives PI3Ks and remodels pathway homeostasis in PTEN-null prostate

Inhibition of BMP2 and BMP4 Represses Barrett’s Esophagus While Enhancing the Regeneration of Squamous Epithelium in Preclinical Models

Eprenetapopt triggers ferroptosis, inhibits NFS1 cysteine desulfurase, and synergizes with serine and glycine dietary restriction

Elevation of fatty acid desaturase 2 in esophageal adenocarcinoma increases polyunsaturated lipids and may exacerbate bile acid‐induced DNA damage

Epithelial de-differentiation triggered by co-ordinate epigenetic inactivation of the EHF and CDX1 transcription factors drives colorectal cancer progression.

Physiological expression of PI3K H1047R mutation reveals its anti-metastatic potential in ErbB2-driven breast cancer

Tumor-Infiltrating Neutrophils after Neoadjuvant Therapy are Associated with Poor Prognosis in Esophageal Cancer.

Molecular and genomic characterisation of a panel of human anal cancer cell lines.

SLC7A11 Is a Superior Determinant of APR-246 (Eprenetapopt) Response than <i>TP53</i> Mutation Status.

Trapping Colorectal Cancer Into a Dead-end

HOXA13 in etiology and oncogenic potential of Barrett’s esophagus

Loss of SMAD4 Is Sufficient to Promote Tumorigenesis in a Model of Dysplastic Barrett's Esophagus.

Transketolase regulates sensitivity to APR-246 in p53-null cells independently of oxidative stress modulation.

Collaborators (15)

Dmitri Mouradov

Thomas Stoll

Sylvain Garciaz

Larry Croft

Sharad Kumar

Pasquale Rescigno

Lochlan Fennell

Sandra Brosda

Diana Stojanovski

Sue Haupt

Gisela Mir Arnau

Richard W Tothill

Michelle M. Hill

Yoon Lim

Lisa A. Mielke

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