Weikang Ma

Research Assistant Professor

Illinois Institute of Technology

United States

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Articles (18)

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2024

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2023

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2022

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2022

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2022

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2022

Collaborators (12)

Jose R Pinto

Professor

University of Florida College of Medicine

UNITED STATES

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Monica Mukherjee

Associate Professor of Medicine

Johns Hopkins University School of Medicine

UNITED STATES

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Jonathan A. Kirk

UNITED STATES

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Thomas Irving

Professor

Illinois Institute of Technology

UNITED STATES

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Roger Craig

UNITED STATES

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Wolfgang A. Linke

GERMANY

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Bert Tanner

Associate Professor

Washington State University

UNITED STATES

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David Kass

Professor, Researcher, Lab Director

Johns Hopkins University

UNITED STATES

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Mohammed Aslam

Duke University

UNITED STATES

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Kyounghwan Lee

Assistant Professor

University of Massachusetts Boston

UNITED STATES

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Sakthivel Sadayappan

Professor

University of Cincinnati

UNITED STATES

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Bradley M Palmer

Assistant Professor

University of Vermont

UNITED STATES

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Weikang Ma

Research Interests

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Articles (18)

Fast myosin binding protein C knockout in skeletal muscle alters length-dependent activation and myofilament structure

The structural and functional effects of myosin regulatory light chain phosphorylation are amplified by increases in sarcomere length and [Ca<sup>2+</sup>].

Differences in thick filament activation in fast rodent skeletal muscle and slow porcine cardiac muscle

Titin-Based Force Modulates Cardiac Thick and Thin Filaments

Myosin-binding protein C regulates the sarcomere lattice and stabilizes the OFF states of myosin heads

Myosin in autoinhibited off state(s), stabilized by mavacamten, can be recruited in response to inotropic interventions

The structural OFF and ON states of myosin can be decoupled from the biochemical super- and disordered-relaxed states

The structural and functional integrities of porcine myocardium are mostly preserved by cryopreservation

Danicamtiv Increases Myosin Recruitment and Alters Cross-Bridge Cycling in Cardiac Muscle

Right Ventricular Sarcomere Contractile Depression and the Role of Thick Filament Activation in Human Heart Failure With Pulmonary Hypertension

Cardiac troponin T N-domain variant destabilizes the actin interface resulting in disturbed myofilament function

The distinctive mechanical and structural signatures of residual force enhancement in myofibers.

Structural OFF/ON transitions of myosin in relaxed porcine myocardium predict calcium-activated force

Disruption of Z-Disc Function Promotes Mechanical Dysfunction in Human Myocardium: Evidence for a Dual Myofilament Modulatory Role by Alpha-Actinin 2

MgADP Promotes Myosin Head Movement toward Actin at Low [Ca<sup>2+</sup>] to Increase Force Production and Ca<sup>2+</sup>-Sensitivity of Contraction in Permeabilized Porcine Myocardial Strips

Cardiac myosin filaments are directly regulated by calcium

Thick filament activation is different in fast‐ and slow‐twitch skeletal muscle

Titin force in muscle cells alters lattice order, thick and thin filament protein formation

Collaborators (12)

Jose R Pinto

Monica Mukherjee

Jonathan A. Kirk

Thomas Irving

Roger Craig

Wolfgang A. Linke

Bert Tanner

David Kass

Mohammed Aslam

Kyounghwan Lee

Sakthivel Sadayappan

Bradley M Palmer

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