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Yoshiaki Suzuki

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Assistant Professor

Nagoya University

Japan

Research Interests

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Professors in Japan Professors in Cell Biology Cell Biology professors in Japan

Recent Grants

Grant: Close

Involvement of inter-organelle Ca2+ network in developement of deseases caused by smooth muscle remoedeling

Open Date: 2019-04-01

Close Date: 2022-03-31

Grant: Close

Pathophysiological functions of ion channels in the chronic inflammatory and fibrotic disorders

Open Date: 2018-10-09

Close Date: 2021-03-31

Grant: Close

Functions of fuctionally silent potassium channels in macrophages

Open Date: 2016-04-01

Close Date: 2018-03-31

Grant: Close

興奮-転写連関による転写遺伝子の特異的選択機構の解明

Open Date: 2016-04-01

Close Date: 2020-03-31

Grant: Close

Molecular pharmacological analyses of calcium-activated ion channels as novel drug targets

Open Date: 2014-04-01

Close Date: 2018-03-31

Articles (24)

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Collaborators (5)

Junko Kurokawa

Professor

Shizuoka University

JAPAN

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Takeshi Akiyoshi

Keio University

JAPAN

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Ali Mobasheri

Professor of Musculoskeletal Biology

University of Oulu

FINLAND

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Hisao Yamamura

Professor

Nagoya University

JAPAN

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Hisakazu Ohtani

Professor

Keio University

JAPAN

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Yoshiaki Suzuki

Research Interests

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Recent Grants

Articles (24)

TMEM16A Ca2+‐activated Cl− channel: Functional interactions with cytoskeletal and membrane‐associated proteins

Luzindole and ramelteon share a melatonin receptor-independent blocking effect on voltage-gated potassium KV4.2 channels

Interleukin-1β downregulates K+ currents through Kv1.6 channels and promotes apoptosis by enhancing Ca2+ influx in chondrocytes

Therapeutic effects of fingolimod through sphingosine-1-phosphate signaling in pulmonary arterial hypertension

Upregulation of T-type voltage-dependent Ca²⁺ channels in activated hepatic stellate cells promotes liver fibrosis in metabolic dysfunction-associated steatohepatitis

Repositioning lidocaine as a TMEM16A Ca2+-activated Cl− channel blocker for the treatment of pulmonary arterial hypertension

Enhanced Yoda1-induced vasoconstriction in pulmonary arterial smooth muscle from monocrotaline-induced pulmonary hypertensive rats

TGF-β1-induced downregulation of the mitochondrial Ca2+ uniporter facilitates the migration of hepatic stellate cells

Microtubule-dependent regulation of TMEM16A-mediated Ca2+-activated Cl− currents in vascular smooth muscle cells

Kir2.1 channels drive macrophage migration through enhancing store-operated Ca2+ entry

Comparative analysis of the single-molecule transport kinetics of OATP1B1 genetic variants

Ca2+ microdomain-based excitation-transcription coupling in cardiac myocytes and vascular smooth muscle cells

Ca2+ microdomains in vascular smooth muscle cells: Roles in vascular tone regulation and hypertension

Caveolin-1 forms a complex with P2X7 receptor and tunes P2X7-mediated ATP signaling in mouse bone marrow-derived macrophages

Mitofusin 1 and 2 differentially regulate mitochondrial function underlying Ca2+ signaling and proliferation in rat aortic smooth muscle cells

Mitofusin 2 positively regulates Ca2+ signaling by tethering the sarcoplasmic reticulum and mitochondria in rat aortic smooth muscle cells

Involvement of small-conductance Ca2+-activated K+ (SKCa2) channels in spontaneous Ca2+ oscillations in rat pinealocytes

A molecular complex of Ca<sub>v</sub>1.2/CaMKK2/CaMK1a in caveolae is responsible for vascular remodeling via excitation-transcription coupling.

SKF96365 activates calcium-sensing receptors in pulmonary arterial smooth muscle cells.

Downregulation of Ca<sup>2+</sup>-Activated Cl<sup>-</sup> Channel TMEM16A Mediated by Angiotensin II in Cirrhotic Portal Hypertensive Mice.

Involvement of TREK1 channels in the proliferation of human hepatic stellate LX-2 cells.

Editorial: Regulatory Mechanisms of Ca<sup>2+</sup> Activated Ion Channels and Their Impact on Physiological/Pathophysiological Function

Comparative analysis of age in monocrotaline-induced pulmonary hypertensive rats.

Hypoxia increases the proliferation of brain capillary endothelial cells via upregulation of TMEM16A Ca<sup>2+</sup>-activated Cl<sup>-</sup> channels.

Collaborators (5)

Junko Kurokawa

Takeshi Akiyoshi

Ali Mobasheri

Hisao Yamamura

Hisakazu Ohtani

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