Yukiko Yoshida

Tokyo Metropolitan Institute of Medical Science

Japan

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Articles (11)

Year:

2025

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2025

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2024

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2024

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2024

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2024

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2024

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2022

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2021

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2021

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2020

Collaborators (13)

Yasushi Saeki

Professor

University of Tokyo

JAPAN

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Fuminori Tokunaga

Professor

Osaka University

JAPAN

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Nozomi Ishii

Assistant Professor

Gunma University

JAPAN

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Noriyuki Matsuda

Professor

Tokyo Medical and Dental University

JAPAN

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Toshiaki Fukushima

Assistant Professor

Tokyo Institute of Technology

JAPAN

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Tsuyoshi Takahashi

Associate Professor

Gunma University

JAPAN

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Tsunehiro Mizushima

Professor

University of Hyogo

JAPAN

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Hirokazu Yagi

Assistant Professor

Nagoya University

JAPAN

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Tony Ly

UNITED KINGDOM

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Kenta Moriwaki

Assistant Professor

Toho University

JAPAN

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Ichiro Matsuo

Gunma University

JAPAN

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Kazuhiro Iwai

JAPAN

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Shigetsugu Hatakeyama

Professor

Hokkaido University Graduate School of Medicine

JAPAN

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Yukiko Yoshida

Research Interests

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Articles (11)

Ubiquitination-activated TAB–TAK1–IKK–NF-κB axis modulates gene expression for cell survival in the lysosomal damage response

Ubiquitination-activated TAB–TAK1–IKK–NF-κB axis modulates gene expression for cell survival in the lysosomal damage response

Structural basis of sugar recognition by SCFFBS2 ubiquitin ligase involved in NGLY1 deficiency

USP8 prevents aberrant NF-κB and Nrf2 activation by counteracting ubiquitin signals from endosomes

Sugar-mediated non-canonical ubiquitination impairs Nrf1/NFE2L1 activation

ELISA-based highly sensitive assay system for the detection of endogenous NGLY1 activity

Ubiquitin-mediated endosomal stress: A novel organelle stress of early endosomes that initiates cellular signaling pathways: USP8 serves as a gatekeeper of ubiquitin-mediated endosomal stress to counteract the activation of cellular signaling pathways.

Luminescence detection of peptide:N-glycanase activity using engineered split inteins

MIND bomb 2 prevents RIPK1 kinase activity-dependent and -independent apoptosis through ubiquitylation of cFLIPL

Loss of peptide:N-glycanase causes proteasome dysfunction mediated by a sugar-recognizing ubiquitin ligase

A substrate-trapping strategy to find E3 ubiquitin ligase substrates identifies Parkin and TRIM28 targets

Collaborators (13)

Yasushi Saeki

Fuminori Tokunaga

Nozomi Ishii

Noriyuki Matsuda

Toshiaki Fukushima

Tsuyoshi Takahashi

Tsunehiro Mizushima

Hirokazu Yagi

Tony Ly

Kenta Moriwaki

Ichiro Matsuo

Kazuhiro Iwai

Shigetsugu Hatakeyama

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