ZHONGJIE FU

Assistant Professor

Boston Children's Hospital/Harvard Medical School

United States

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Dr. Zhongjie Fu is an Assistant Professor at Boston Children's Hospital and Harvard Medical School in the United States. His research focuses on retinal diseases and angiogenesis, with recent publications exploring mechanisms of choroidal neovascularization and therapeutic interventions. Notable studies include the effects of nutrient supplementation on retinal dysfunction and the role of specific receptors in retinal vascular health.

Articles (16)

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2025

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2025

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2025

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2023

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2022

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2022

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2022

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2022

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2021

Collaborators (11)

Deokho Lee

Research Professor

Kagawa Nutrition University

SOUTH KOREA

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Martha Field

Assistant Professor

Cornell University

UNITED STATES

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Lois Smith

Professor in Ophthalmology

Harvard Medical School

UNITED STATES

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Yohei Tomita

Senior assistant professor

Keio University School of Medicine

JAPAN

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Anders Nilsson

Göteborgs Universitet

SWEDEN

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Timothy Hla

Patricia Donahoe Professor, Harvard Medical School

Boston Children's Hospital/Harvard Medical School

UNITED STATES

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Ayumi Usui-Ouchi

Juntendo University

JAPAN

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Edward Bull

Icahn School of Medicine at Mount Sinai

UNITED STATES

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Ann Hellström

Professor

SWEDEN

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Richard Proia

National Institutes of Health

UNITED STATES

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Amy O'Connell

Assistant Professor

Harvard Medical School

UNITED STATES

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ZHONGJIE FU

Research Interests

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About

Articles (16)

Choroidal Neovascularization Is Suppressed With Activation of TREM2 in Mononuclear Phagocytes—Brief Report

ApoM-bound S1P acts via endothelial S1PR1 to suppress choroidal neovascularization and vascular leakage.

Serine supplementation suppresses hypoxia-induced pathological retinal angiogenesis.

Nutrient supplementation mitigates retinal dysfunction in Acox1 knockout mice with impaired peroxisomal fatty acid oxidation.

Folic Acid Supplementation Inhibits Proliferative Retinopathy of Prematurity

Therapeutic Effects of Anti-Inflammatory and Anti-Oxidant Nutritional Supplementation in Retinal Ischemic Diseases

Therapeutic activation of endothelial sphingosine‐1‐phosphate receptor 1 by chaperone‐bound S1P suppresses proliferative retinal neovascularization

In vivo noninvasive mitochondrial redox assessment of the optic nerve head to predict disease

Ectopic Rod Photoreceptor Development in Mice with Genetic Deficiency of WNT2B

Retinopathy of prematurity: Metabolic risk factors

Murine endothelial serine palmitoyltransferase 1 (SPTLC1) is required for vascular development and systemic sphingolipid homeostasis

Omega-3/Omega-6 Long-Chain Fatty Acid Imbalance in Phase I Retinopathy of Prematurity.

Amino acid transporter SLC38A5 regulates developmental and pathological retinal angiogenesis

REV-ERBα regulates age-related and oxidative stress-induced degeneration in retinal pigment epithelium via NRF2

Retinal Disease and Metabolism

Müller glial responses compensate for degenerating photoreceptors in retinitis pigmentosa.

Collaborators (11)

Deokho Lee

Martha Field

Lois Smith

Yohei Tomita

Anders Nilsson

Timothy Hla

Ayumi Usui-Ouchi

Edward Bull

Ann Hellström

Richard Proia

Amy O'Connell

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