Martha Field

Assistant Professor

Cornell University

United States

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Martha Field is an Assistant Professor at Cornell University, USA. Her research areas include the impacts of dietary supplements on pregnancy outcomes, the role of artificial intelligence in food and nutrition, and the effects of nutrient status on mitochondrial DNA integrity and obesity risks. She has contributed to recent articles examining the implications of folic acid and riboflavin on health outcomes in animal models.

Articles (18)

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2025

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2020

Collaborators (13)

Deokho Lee

Research Professor

Kagawa Nutrition University

SOUTH KOREA

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Joanna Fiddler

Assistant Professor

University of Arkansas at Fayetteville

UNITED STATES

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Nathaniel Vacanti

Assistant Professor of Molecular Nutrition

Cornell University

UNITED STATES

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David Klurfeld

Adjunct Professor

Indiana State University

UNITED STATES

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Peter Kühnen

GERMANY

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Elsa Leitão

Universitätsklinikum Essen Institut für Humangenetik

GERMANY

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Christian Metallo

Professor

Salk Institute for Biological Studies

UNITED STATES

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Richard Finnell

Professor

Baylor College of Medicine

UNITED STATES

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Yvonne Lamers

Associate Professor; Canada Research Chair

The University of British Columbia

CANADA

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Benedikt Obermayer

Berlin Institute of Health at Charité - Universitätsmedizin Berlin

GERMANY

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Valeria Fernandez Vallone

Charité - Universitätsmedizin Berlin

GERMANY

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Alice H Lichtenstein

Tufts University

UNITED STATES

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ZHONGJIE FU

Assistant Professor

Boston Children's Hospital/Harvard Medical School

UNITED STATES

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Martha Field

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Articles (18)

Folic Acid Supplementation Inhibits Proliferative Retinopathy of Prematurity

Systematic review of the impact of ginger extract and alpinetin on pregnancy outcomes in animal models.

Vitamin B12 status and folic acid supplementation influence mitochondrial heteroplasmy levels in mice

Artificial intelligence in food and nutrition evidence: The challenges and opportunities

Excess Folic Acid Exposure Increases Uracil Misincorporation into DNA in a Tissue-Specific Manner in a Mouse Model of Reduced Methionine Synthase Expression

Reduced methionine synthase expression results in uracil accumulation in mitochondrial DNA and impaired oxidative capacity

Early-set POMC methylation variability is accompanied by increased risk for obesity and is addressable by MC4R agonist treatment

C2C12 Muscle Myotubes, but not Kidney Proximal Tubule HK-2 Cells, Elevate Erythritol Synthesis in Response to Oxidative Stress

Unrecognized riboflavin deficiency and evidence for cascading effects on vitamin B-6 status

Scanning the evidence: process and lessons learned from an evidence scan of riboflavin to inform decisions on updating the riboflavin dietary reference intakes

Pyruvate Kinase M2 Supports Muscle Progenitor Cell Proliferation but Is Dispensable for Skeletal Muscle Regeneration after Injury

Reduced Shmt2 Expression Impairs Mitochondrial Folate Accumulation and Respiration, and Leads to Uracil Accumulation in Mouse Mitochondrial DNA

Chronic Dietary Erythritol Exposure Elevates Plasma Erythritol Concentration in Mice but Does Not Cause Weight Gain or Modify Glucose Homeostasis

Knowledge gaps in understanding the metabolic and clinical effects of excess folates/folic acid: a summary, and perspectives, from an NIH workshop

The Roles of Mitochondrial Folate Metabolism in Supporting Mitochondrial DNA Synthesis, Oxidative Phosphorylation, and Cellular Function

Wheat flour fortification with iron for reducing anaemia and improving iron status in populations.

Mammalian metabolism of erythritol: a predictive biomarker of metabolic dysfunction.

Emerging Concepts in Nutrient Needs

Collaborators (13)

Deokho Lee

Joanna Fiddler

Nathaniel Vacanti

David Klurfeld

Peter Kühnen

Elsa Leitão

Christian Metallo

Richard Finnell

Yvonne Lamers

Benedikt Obermayer

Valeria Fernandez Vallone

Alice H Lichtenstein

ZHONGJIE FU

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