Amila Suraweera

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Research Fellow/Lecturer

Queensland University of Technology

Australia

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About

Amila Suraweera is a Research Fellow and Lecturer at Queensland University of Technology in Australia. His research focuses on the roles of various proteins in cancer cell proliferation, survival, and DNA repair mechanisms, with recent publications addressing topics such as triple negative breast cancer, epigenetic drugs in therapy, and the regulation of DNA double-strand break repair pathways. He contributes to advancements in cancer therapeutics by exploring targeted strategies that influence cellular metabolism and repair processes.

Recent Grants

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Development of a novel therapeutic for the treatment of non-small cell lung cancer

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Articles (13)

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2025

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2025

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2023

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2023

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2021

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2021

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2021

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2021

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2020

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2020

Collaborators (7)

James AL Brown

University of Limerick

IRELAND

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Mark Adams

Queensland University of Technology

AUSTRALIA

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Emma Bolderson

Senior Research Fellow / Senior Lecturer

Queensland University of Technology

AUSTRALIA

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Pascal Duijf

Associate Professor

University of South Australia

AUSTRALIA

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Kienan Savage

Reader/Group Leader

The Queen's University Belfast

UNITED KINGDOM

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Kenneth O'Byrne

Queensland University of Technology

AUSTRALIA

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Derek Richard

Queensland University of Technology

AUSTRALIA

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Amila Suraweera

Research Interests

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About

Recent Grants

Articles (13)

The inner nuclear membrane protein, Banf1, has an essential role in triple negative breast cancer cell proliferation and survival

Epigenetic drugs in cancer therapy

The fructose-bisphosphate, Aldolase A (ALDOA), facilitates DNA-PKcs and ATM kinase activity to regulate DNA double-strand break repair

Mono-phosphorylation at Ser4 of barrier-to-autointegration factor (Banf1) significantly reduces its DNA binding capability by inducing critical changes in its local conformation and DNA binding surface

Targeting the COMMD4–H2B protein complex in lung cancer

Editorial: Cancer Therapeutics: Targeting DNA Repair Pathways

Epigenetic Mechanisms in DNA Double Strand Break Repair: A Clinical Review

Cell Metabolism and DNA Repair Pathways: Implications for Cancer Therapy

Barrier-To-Autointegration-factor (Banf1) modulates DNA double-strand break repair pathway choice via regulation of DNA-dependent kinase (DNA-PK) activity

COMMD4 functions with the histone H2A-H2B dimer for the timely repair of DNA double-strand breaks

COMMD1, from the Repair of DNA Double Strand Breaks, to a Novel Anti-Cancer Therapeutic Target

Defining COMMD4 as an anti-cancer therapeutic target and prognostic factor in non-small cell lung cancer

COMMD4 Functions with the Histone H2A-H2B Dimer for the Timely Repair of DNA Double Strand Breaks

Collaborators (7)

James AL Brown

Mark Adams

Emma Bolderson

Pascal Duijf

Kienan Savage

Kenneth O'Byrne

Derek Richard

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