Arsalan Yousuf

Lecturer

University of Sydney

Australia

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Arsalan Yousuf is a Lecturer at the University of Sydney, Australia. His research focuses on the applications of artificial intelligence in education and the pharmacological modulation of nicotinic acetylcholine receptors. Recent publications explore the impact of generative AI on student assessments and investigate the mechanisms of various compounds affecting neuronal excitability and pain modulation.

Articles (9)

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2025

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2025

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2024

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2024

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2023

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2022

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2021

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2020

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2020

Collaborators (1)

David John Adams

Distinguished Professor

University of Wollongong

AUSTRALIA

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Arsalan Yousuf

Research Interests

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Articles (9)

AI in action: Changes to student perceptions when using generative artificial intelligence for the creation of a multimedia project-based assessment.

GABAB Receptor Modulation of Membrane Excitability in Human Pluripotent Stem Cell‐Derived Sensory Neurons by Baclofen and α‐Conotoxin Vc1.1

Tabernanthalog and ibogainalog inhibit the α7 and α9α10 nicotinic acetylcholine receptors via different mechanisms and with higher potency than the GABA<sub>A</sub> receptor and Ca<sub>V</sub>2.2 channel.

Dual Antagonism of α9α10 nAChR and GABA<sub>B</sub> Receptor-Coupled Ca<sub>V</sub>2.2 Channels by an Analgesic αO-Conotoxin Analogue.

The Antinociceptive Activity of (E)-3-(thiophen-2-yl)-N-(p-tolyl)acrylamide in Mice Is Reduced by (E)-3-(furan-2-yl)-N-methyl-N-(p-tolyl)acrylamide Through Opposing Modulatory Mechanisms at the α7 Nicotinic Acetylcholine Receptor.

A 4/8 Subtype α-Conotoxin Vt1.27 Inhibits N-Type Calcium Channels With Potent Anti-Allodynic Effect

ɑO‐Conotoxin GeXIVA isomers modulate N‐type calcium (Ca V 2.2) channels and inwardly‐rectifying potassium (GIRK) channels via GABA B receptor activation

(<i>E</i>)-3-Furan-2-yl-<i>N</i>-<i>p</i>-tolyl-acrylamide and its Derivative DM489 Decrease Neuropathic Pain in Mice Predominantly by α7 Nicotinic Acetylcholine Receptor Potentiation

Coronaridine congeners decrease neuropathic pain in mice and inhibit α9α10 nicotinic acetylcholine receptors and Ca<inf>V</inf>2.2 channels

Collaborators (1)

David John Adams

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