Donald Bers

Distinguished Professor & Chair

University of California Davis School of Medicine

United States

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About

Donald Bers is a Distinguished Professor & Chair at the University of California Davis School of Medicine, USA. His research areas encompass molecular and cellular neurocardiology, cardiac physiology, and the mechanisms underlying heart disease. Recent articles authored by Professor Bers explore topics such as the effects of hyperglycaemia on cardiac cells, sex-dependent cardiac responses in obesity-related heart failure, and innovative techniques for analyzing calcium dynamics in muscle receptors.

Articles (17)

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2025

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2022

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2022

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2022

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2022

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2022

Collaborators (16)

Eleonora Grandi

University of California, Davis

UNITED STATES

Donald Bers

Research Interests

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Articles (17)

Hyperglycaemia‐induced reactive oxygen species production in cardiac ventricular myocytes differs among mammals

Kinetics and mapping of Ca-driven calmodulin conformations on skeletal and cardiac muscle ryanodine receptors

Cardiac Na-Ca exchanger “tunes-out” at high intracellular [Na]

Buffering the Heart Against Atrial Fibrillation

Molecular and cellular neurocardiology in heart disease

Differential sex-dependent susceptibility to diastolic dysfunction and arrhythmia in cardiomyocytes from obese diabetic heart failure with preserved ejection fraction model

Molecular Mechanism of a FRET Biosensor for the Cardiac Ryanodine Receptor Pathologically Leaky State

Diversity of cells and signals in the cardiovascular system

SparkMaster 2: A New Software for Automatic Analysis of Calcium Spark Data

New cardiac targets for empagliflozin: O-GlcNAcylation, CaMKII, and calcium handling

Whole-heart multiparametric optical imaging reveals sex-dependent heterogeneity in cAMP signaling and repolarization kinetics

Nitric Oxide Modulates Ca 2+ Leak and Arrhythmias via S-Nitrosylation of CaMKII

Diabetes and Excess Aldosterone Promote Heart Failure With Preserved Ejection Fraction

Cardiac Ca2+ current is ‘Rad’-ically altered by adrenergic activation

Nitrosylation of cardiac CaMKII at Cys290 mediates mechanical afterload‐induced increases in Ca2+ transient and Ca2+ sparks

Protein Kinase D1 Regulates Cardiac Hypertrophy, Potassium Channel Remodeling, and Arrhythmias in Heart Failure

Subcellular Propagation of Cardiomyocyte β-Adrenergic Activation of Calcium Uptake Involves Internal β-Receptors and AKAP7

Collaborators (16)

Eleonora Grandi

David Jones

David D. Thomas

Nikki Gillum Posnack

Madeline Nieves-Cintron

Dan Li

Matthew Kay

Ye Chen-Izu

Julie Bossuyt

Dobromir Dobrev

Crystal M. Ripplinger

David Mendelowitz

Răzvan L. Cornea

Osama Harraz

Beth Habecker

Rose Dixon

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