Rose Dixon

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Assistant Professor

University of California, Davis

United States

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About

Dr. Rose Dixon is an Assistant Professor at the University of California Davis, USA. Her research focuses on cardiac physiology, cellular signaling, and excitation-contraction coupling, as evidenced by her recent articles on membrane-curving adaptor proteins and cardiac Ca V 1.2 regulation. She contributes to understanding cardiac dysfunction and cellular mechanisms, particularly in relation to ATP dynamics and the implications of cytoskeletal disarray in heart health.

Recent Grants

Grant: Close

Molecular choreography of CaV1.2 channels in the aging myocardium

Open Date: 2019-08-01

Close Date: 2024-04-01

Grant: Close

Cardiac EC coupling: Many L-type Ca2+ channels make sparks work.

Open Date: 2015-07-01

Close Date: 2019-06-30

Articles (14)

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2025

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2023

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2021

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2021

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2021

Collaborators (16)

Eleonora Grandi

University of California, Davis

UNITED STATES

Rose Dixon

Research Interests

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About

Recent Grants

Articles (14)

Bend it like BIN1: how a membrane-curving adaptor protein shapes cardiac physiology

14‐3‐3 proteins: Regulators of cardiac excitation–contraction coupling and stress responses

14-3-3 promotes sarcolemmal expression of cardiac Ca V 1.2 and nucleates isoproterenol-triggered channel superclustering

The Piezo channel is a mechano-sensitive complex component in the mammalian inner ear hair cell.

Publisher Correction: BIN1 knockdown rescues systolic dysfunction in aging male mouse hearts

BIN1 knockdown rescues systolic dysfunction in aging male mouse hearts

Fueling the heartbeat: Dynamic regulation of intracellular ATP during excitation–contraction coupling in ventricular myocytes

Diversity of cells and signals in the cardiovascular system

Acute phosphatidylinositol 4,5 bisphosphate depletion destabilizes sarcolemmal expression of cardiac L-type Ca 2+ channel Ca V 1.2

Cytoskeletal disarray increases arrhythmogenic vulnerability during sympathetic stimulation in a model of hypertrophic cardiomyopathy

NPC1-dependent alterations in KV2.1–CaV1.2 nanodomains drive neuronal death in models of Niemann-Pick Type C disease

IP 3 R-driven increases in mitochondrial Ca 2+ promote neuronal death in NPC disease

NPC1 regulates the distribution of phosphatidylinositol 4‐kinases at Golgi and lysosomal membranes

β-Adrenergic control of sarcolemmal Ca V 1.2 abundance by small GTPase Rab proteins

Collaborators (16)

Eleonora Grandi

David Jones

Nikki Gillum Posnack

Livia Hool

Donald Bers

Mary Horne

Bernd Fritzsch

Henrietta Cserne Szappanos

Dobromir Dobrev

Padmini Sirish

Eamonn Dickson

Phung N Thai

Johannes W Hell

ryan woltz

Osama Harraz

James S. Trimmer

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