Eugenio Barone

Associate Professor

Sapienza University of Rome

Italy

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Eugenio Barone is an Associate Professor at Sapienza University of Rome, Italy. His research primarily focuses on the relationship between insulin signaling, mitochondrial dysfunction, and neurodegeneration, with particular attention to Alzheimer's disease and Down syndrome. Recent articles include studies on insulin resistance and its role in neuroinflammation as well as oxidative stress in metabolic disorders.

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Collaborators (13)

Vito Pesce

University of Bari Aldo Moro

ITALY

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Federico Marini

Full professor

Università degli Studi di Roma La Sapienza

ITALY

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Eleonora Palma

Full Professor

Sapienza University of Rome

ITALY

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Alberto Costa

Professor

Case Western Reserve University

UNITED STATES

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Fabio Di Domenico

Full Professor

Sapienza University of Rome

ITALY

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Maria Gisella Cavallo

Sapienza University

ITALY

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Claudio GRASSI

Università Cattolica del Sacro Cuore -Fondazione Policlinico Universitario Agostino Gemelli - IRCCS

ITALY

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Ilaria Barchetta

Assistant Professor (Tenure track)

Sapienza University of Rome

ITALY

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Ana Lloret

Full Professor

University of Valencia

SPAIN

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Andre Strydom

King’s College London

UNITED KINGDOM

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Gabriele Ruffolo

Sapienza University of Rome

ITALY

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Li Chan

Professor of Molecular Endocrinology and Metabolism/Honorary Consultant in Paediatric Endocrinology

UNITED KINGDOM

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Cristian RIPOLI

Associate Professor

ITALY

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Eugenio Barone

Research Interests

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Articles (20)

Imagine, Discover, Inspire: Proceedings of the 4th International Conference of the Trisomy 21 Research Society.

Biliverdin reductase-A is a key modulator in insulin signaling and metabolism

Insulin signaling in microglia: A metabolic switch controlling neuroinflammation and amyloid pathology in Alzheimer’s disease

Metabolic breakdown: Linking insulin resistance and mitochondrial dysfunction to neurodegeneration in Alzheimer’s disease

Metabolic breakdown: Linking insulin resistance and mitochondrial dysfunction to neurodegeneration in Alzheimer’s disease

Insulin resistance: fueling oxidative stress and neurodegeneration

Altered mitochondrial unfolded protein response and protein quality control promote oxidative distress in down syndrome brain

Corrigendum to “Altered mitochondrial unfolded protein response and protein quality control promote oxidative distress in down syndrome brain” [Free Radic. Biol. Med. 227 (2025) 80-93, (S0891584924010773), (10.1016/j.freeradbiomed.2024.11.043)]

Pharmacokinetics and pharmacodynamics of intranasal insulin in a rat brain

Aging exacerbates oxidative stress and liver fibrosis in an animal model of Down Syndrome

Dynamic Changes of BVRA Protein Levels Occur in Response to Insulin: A Pilot Study in Humans

A Bioengineering Strategy to Control ADAM10 Activity in Living Cells

Intranasal Administration of KYCCSRK Peptide Rescues Brain Insulin Signaling Activation and Reduces Alzheimer’s Disease-Like Neuropathology in a Mouse Model for Down Syndrome

Role of Biliverdin Reductase A in the Regulation of Insulin Signaling in Metabolic and Neurodegenerative Diseases: An Update

Role of Biliverdin Reductase A in the Regulation of Insulin Signaling in Metabolic and Neurodegenerative Diseases: An Update

Aberrant crosstalk between insulin signaling and mTOR in young Down syndrome individuals revealed by neuronal-derived extracellular vesicles

Aberrant protein networks in Alzheimer disease

Brain insulin resistance: an early risk factor for Alzheimer's disease development in Down syndrome

CAPE and its synthetic derivative VP961 restore BACH1/NRF2 axis in Down Syndrome

Insulin Signaling Regulates Mitochondrial Bioenergetics in the Brain: Insight for the Development of Neurodegenerative Diseases

Collaborators (13)

Vito Pesce

Federico Marini

Eleonora Palma

Alberto Costa

Fabio Di Domenico

Maria Gisella Cavallo

Claudio GRASSI

Ilaria Barchetta

Ana Lloret

Andre Strydom

Gabriele Ruffolo

Li Chan

Cristian RIPOLI

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