Gyorgy Hajnoczky

Professor

Thomas Jefferson University

United States

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Gyorgy Hajnoczky is a Professor at Thomas Jefferson University in the United States. His research primarily focuses on organelle membrane contact sites, mitochondrial function, and calcium signaling. Recent publications highlight his work on the roles of mitochondria in intracellular calcium dynamics and the implications of mitochondrial morphology on metabolic processes.

Articles (13)

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2025

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2025

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2024

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2023

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2023

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2023

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2023

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2023

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2022

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2022

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2022

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2022

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2021

Collaborators (8)

Benoît Kornmann

Associate Professor

University of Oxford

UNITED KINGDOM

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Pooja Jadiya

Assistant Professor

Wake Forest Baptist Medical Center

UNITED STATES

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Hyun-Woo Rhee

Associate Professor

Seoul National University

SOUTH KOREA

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Zachary Schug

Assistant Professor

The Wistar Institute

UNITED STATES

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John W. Elrod

Lewis Katz School of Medicine at Temple University

UNITED STATES

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Dhanendra Tomar

Assistant Professor

Wake Forest Baptist Medical Center

UNITED STATES

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Péter Várnai

hungarian research network and semmelweis university

HUNGARY

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Mélanie Paillard

Associate Professor - Chargée de Recherche Hors Classe (CRHC)

INSERM Délégation Régionale Rhône-Alpes

FRANCE

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Gyorgy Hajnoczky

Research Interests

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Contact this professor

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Articles (13)

Key challenges and recommendations for defining organelle membrane contact sites

The roles of mitochondria in global and local intracellular calcium signalling

Supralinear Dependence of the IP3 Receptor-to-Mitochondria Local Ca2+ Transfer on the Endoplasmic Reticulum Ca2+ Loading

MICU1 regulates mitochondrial cristae structure and function independently of the mitochondrial Ca 2+ uniporter channel

Answer to Gerber et al. “Autosomal recessive pathogenic MSTO1 variants in hereditary optic atrophy”

Fuel oxidation under the control of mitochondrial shape and dynamics

MICU1 controls the sensitivity of the mitochondrial Ca2+ uniporter to activators and inhibitors

OPA1 disease-causing mutants have domain-specific effects on mitochondrial ultrastructure and fusion

Capture at the ER-mitochondrial contacts licenses IP3 receptors to stimulate local Ca2+ transfer and oxidative metabolism

Altered composition of the mitochondrial Ca2+uniporter in the failing human heart

Uncontrolled mitochondrial calcium uptake underlies the pathogenesis of neurodegeneration in MICU1-deficient mice and patients

Fluorescence imaging detection of nanodomain redox signaling events at organellar contacts

Metabolic adaptation to the chronic loss of Ca<sup>2+</sup> signaling induced by KO of IP<sub>3</sub> receptors or the mitochondrial Ca<sup>2+</sup> uniporter.

Collaborators (8)

Benoît Kornmann

Pooja Jadiya

Hyun-Woo Rhee

Zachary Schug

John W. Elrod

Dhanendra Tomar

Péter Várnai

Mélanie Paillard

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