Katharina Günther

University of Innsbruck

Austria

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Articles (9)

Year:

2024

Year:

2024

Year:

2024

Year:

2022

Year:

2022

Year:

2021

Year:

2021

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2020

Year:

2020

Collaborators (8)

Michaela Defrancesco

Medical University of Innsbruck

AUSTRIA

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Marja Koskuvi

University of Helsinki

FINLAND

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Francesca Finotello

University of Innsbruck

AUSTRIA

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Andreas G Chiocchetti

Prof

Goethe University Frankfurt

GERMANY

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Sarah Kittel-Schneider

University Hospital Frankfurt

GERMANY

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Frank Edenhofer

Prof., Head Dpt. Genomics, Stem Cell Biology & Regenerative Medicine

University of Innsbruck

AUSTRIA

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Marta Suarez Cubero

University of Innsbruck

AUSTRIA

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Andreas Till

Lecturer & Group Leader

Rheinische Friedrich-Wilhelms-Universität Bonn

GERMANY

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Katharina Günther

Research Interests

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Articles (9)

Single-cell Profiling of Reprogrammed Human Neural Stem Cells Unveils High Similarity to Neural Progenitors in the Developing Central Nervous System

Integrative metabolomics-genomics analysis identifies key networks in a stem cell-based model of schizophrenia

Small fibre neuropathy in Fabry disease: a human-derived neuronal <i>in vitro</i> disease model and pilot data

<scp>ADHD‐associated PARK2</scp> copy number variants: A pilot study on gene expression and effects of supplementary deprivation in patient‐derived cell lines

Generation of the human induced pluripotent stem cell line (IBKMOLi002-A) from PBMCs of a patient carrying the heterozygous L271H mutation of the voltage-gated calcium channel subunit Cav1.3-encoding CACNA1D gene

CRISPR/Cas9-edited PKP2 knock-out (JMUi001-A-2) and DSG2 knock-out (JMUi001-A-3) iPSC lines as an isogenic human model system for arrhythmogenic cardiomyopathy (ACM).

Serotonin-specific neurons differentiated from human iPSCs form distinct subtypes with synaptic protein assembly.

Energy Metabolism Disturbances in Cell Models of PARK2 CNV Carriers with ADHD

Generation of two patient-derived iPSC lines from siblings (LIBUCi001-A and LIBUCi002-A) and a genetically modified iPSC line (JMUi001-A-1) to mimic dilated cardiomyopathy with ataxia (DCMA) caused by a homozygous DNAJC19 mutation

Collaborators (8)

Michaela Defrancesco

Marja Koskuvi

Francesca Finotello

Andreas G Chiocchetti

Sarah Kittel-Schneider

Frank Edenhofer

Marta Suarez Cubero

Andreas Till

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