Kyung Hong

Associate Professor

Western New England University

United States

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Kyung Hong is an Associate Professor at Western New England University in the United States. His research primarily focuses on the cellular and molecular mechanisms of metabolic disorders, cancer biology, and lipid homeostasis, as evidenced by his recent articles on the roles of heterocyclic amines and the N-acetyltransferase 2 gene. Additionally, he explores therapeutic strategies related to the unfolded protein response in inflammatory bowel disease.

Articles (12)

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2024

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2024

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2023

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2023

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2023

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2023

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2023

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2022

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2022

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2022

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2022

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2022

Collaborators (2)

Nobuyuki Matoba

Professor

University of Louisville School of Medicine

UNITED STATES

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David Hein

Professor and Endowed Chair

University of Louisville

UNITED STATES

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Kyung Hong

Research Interests

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Articles (12)

Heterocyclic Amines Disrupt Lipid Homeostasis in Cryopreserved Human Hepatocytes.

Human CKAP2L shows a cell cycle‐dependent expression pattern and exhibits microtubule‐stabilizing properties

The Unfolded Protein Response and Its Implications for Novel Therapeutic Strategies in Inflammatory Bowel Disease.

Induction of glucose production by heterocyclic amines is dependent on N-acetyltransferase 2 genetic polymorphism in cryopreserved human hepatocytes.

N-acetyltransferase 2 haplotype modifies risks for both dyslipidemia and urinary bladder cancer.

Non-coding and intergenic genetic variants of human arylamine <i>N</i>-acetyltransferase 2 (NAT2) gene are associated with differential plasma lipid and cholesterol levels and cardiometabolic disorders.

Heterocyclic amines reduce insulin-induced AKT phosphorylation and induce gluconeogenic gene expression in human hepatocytes.

Transcriptional Regulation of Human Arylamine N-Acetyltransferase 2 Gene by Glucose and Insulin in Liver Cancer Cell Lines.

Upregulation of cytidine deaminase in NAT1 knockout breast cancer cells.

Dataset for proteomic analysis of arylamine <i>N</i>-acetyltransferase 1 knockout MDA-MB-231 breast cancer cells.

Proteomic analysis of arylamine <i>N</i>-acetyltransferase 1 knockout breast cancer cells: Implications in immune evasion and mitochondrial biogenesis.

Deletion of arylamine N-acetyltransferase 1 in MDA-MB-231 human breast cancer cells reduces primary and secondary tumor growth in vivo with no significant effects on metastasis.

Collaborators (2)

Nobuyuki Matoba

David Hein

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