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Maicon Landim-Vieira

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Research Assistant Professor

Illinois Institute of Technology

United States

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Deciphering the structural and molecular processes of myofilament remodeling in phenotype-rescued cardiomyopathy models

Open Date: 2022-01-01

Close Date: 2022-12-18

Articles (17)

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Collaborators (13)

Weikang Ma

Assistant Professor

Illinois Institute of Technology

UNITED STATES

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Taejeong Song

Assistant Professor

University of Arizona

UNITED STATES

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Jose R Pinto

Professor

University of Florida College of Medicine

UNITED STATES

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Mark Ranek

Assistant Professor

Johns Hopkins University

UNITED STATES

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Emily Ehle

Florida State University

UNITED STATES

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Onur Kanisicak

Associate Professor

Ohio State University

UNITED STATES

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Thomas Irving

Professor

Illinois Institute of Technology

UNITED STATES

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Ann Centner

University of Tennessee Health Science Center

UNITED STATES

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Andrew Landstrom

Professor

Duke University School of Medicine

UNITED STATES

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Jerome Irianto

Assistant Professor

University of Florida College of Medicine

UNITED STATES

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Bjorn Knollmann

Professor

Vanderbilt University Medical Center

UNITED STATES

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P. Bryant Chase

Assoc/Full Professor

Florida State University

UNITED STATES

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Joseph Chalovich

Distinguished Professor

East Carolina University

UNITED STATES

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Maicon Landim-Vieira

Research Interests

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Recent Grants

Articles (17)

Desmoglein-2 deficiency drives mitochondrial morphological remodeling in cardiomyocytes

Cardiac Troponin C E135A Variant Impairs Myofilament Response to PKA Phosphorylation and Is Associated With Autosomal Dominant Dilated Cardiomyopathy With Diastolic Dysfunction

ATP directly modulates thick filament structure and function in porcine myocardium

Phenotype specific nuclear lamina remodeling in hiPSC derived cardiomyocytes bearing <i>TNNT2</i> sarcomeric variants.

The role of the troponin T interactions with actin in regulation of cardiac thin filament revealed by the troponin T pathogenic variant Ile79Asn

The role of mechanosignaling in the control of myocardial mass

High-Fat Diet Augments Myocardial Inflammation and Cardiac Dysfunction in Arrhythmogenic Cardiomyopathy

Can evolution-based studies inform modern medicine?

NFĸB signaling drives myocardial injury via CCR2+ macrophages in a preclinical model of arrhythmogenic cardiomyopathy

Cardiac troponin T N-domain variant destabilizes the actin interface resulting in disturbed myofilament function

Negative Charges Introduced Near the IT Helix of Cardiac Troponin T Stabilize the Active State of Actin Filaments

Nucleus Mechanosensing in Cardiomyocytes

Danicamtiv Recruits Myosin Motors to Aid the Failing Heart

Etiology of genetic muscle disorders induced by mutations in fast and slow skeletal MyBP-C paralogs

Disruption of Z-Disc Function Promotes Mechanical Dysfunction in Human Myocardium: Evidence for a Dual Myofilament Modulatory Role by Alpha-Actinin 2

Chronic exposure to mercury increases arrhythmia and mortality post-acute myocardial infarction in rats

Platelet activation and endothelial dysfunction biomarkers in acute coronary syndrome: the impact of PCSK9 inhibition

Collaborators (13)

Weikang Ma

Taejeong Song

Jose R Pinto

Mark Ranek

Emily Ehle

Onur Kanisicak

Thomas Irving

Ann Centner

Andrew Landstrom

Jerome Irianto

Bjorn Knollmann

P. Bryant Chase

Joseph Chalovich

Make your next move.

Maicon Landim-Vieira

Research Interests

Make your next move.

Explore related searches

Recent Grants

Articles (17)

Desmoglein-2 deficiency drives mitochondrial morphological remodeling in cardiomyocytes

Cardiac Troponin C E135A Variant Impairs Myofilament Response to PKA Phosphorylation and Is Associated With Autosomal Dominant Dilated Cardiomyopathy With Diastolic Dysfunction

ATP directly modulates thick filament structure and function in porcine myocardium

Phenotype specific nuclear lamina remodeling in hiPSC derived cardiomyocytes bearing &lt;i&gt;TNNT2&lt;/i&gt; sarcomeric variants.

The role of the troponin T interactions with actin in regulation of cardiac thin filament revealed by the troponin T pathogenic variant Ile79Asn

The role of mechanosignaling in the control of myocardial mass

High-Fat Diet Augments Myocardial Inflammation and Cardiac Dysfunction in Arrhythmogenic Cardiomyopathy

Can evolution-based studies inform modern medicine?

NFĸB signaling drives myocardial injury via CCR2+ macrophages in a preclinical model of arrhythmogenic cardiomyopathy

Cardiac troponin T N-domain variant destabilizes the actin interface resulting in disturbed myofilament function

Negative Charges Introduced Near the IT Helix of Cardiac Troponin T Stabilize the Active State of Actin Filaments

Nucleus Mechanosensing in Cardiomyocytes

Danicamtiv Recruits Myosin Motors to Aid the Failing Heart

Etiology of genetic muscle disorders induced by mutations in fast and slow skeletal MyBP-C paralogs

Disruption of Z-Disc Function Promotes Mechanical Dysfunction in Human Myocardium: Evidence for a Dual Myofilament Modulatory Role by Alpha-Actinin 2

Chronic exposure to mercury increases arrhythmia and mortality post-acute myocardial infarction in rats

Platelet activation and endothelial dysfunction biomarkers in acute coronary syndrome: the impact of PCSK9 inhibition

Collaborators (13)

Weikang Ma

Taejeong Song

Jose R Pinto

Mark Ranek

Emily Ehle

Onur Kanisicak

Thomas Irving

Ann Centner

Andrew Landstrom

Jerome Irianto

Bjorn Knollmann

P. Bryant Chase

Joseph Chalovich

Make your next move.

Phenotype specific nuclear lamina remodeling in hiPSC derived cardiomyocytes bearing <i>TNNT2</i> sarcomeric variants.