Michiel van Gent

Assistant Professor

Erasmus MC

Netherlands

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About

Michiel van Gent is an Assistant Professor at Erasmus MC in the Netherlands. His research focuses on the dynamics of virus infections, particularly herpesviruses and their interactions with host cellular mechanisms. Recent publications have explored topics such as the neurovirulence of various viruses and the pathways through which they evade innate immunity.

Articles (13)

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2025

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2024

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2023

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2022

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2021

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2021

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2021

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2021

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2021

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2020

Collaborators (10)

Jos A. G. Van Strijp

Professor

University Medical Center Groningen

NETHERLANDS

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Nir Drayman

Assistant Professor

University of California, Irvine

UNITED STATES

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Tomasz Prajsnar

Jagiellonian University

POLAND

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Michael Boettcher

Juniorprofessor

Martin Luther University Halle-Wittenberg

GERMANY

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Stephen A. Renshaw

University of Sheffield

UNITED KINGDOM

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Werner J.D. Ouwendijk

Erasmus University Rotterdam

NETHERLANDS

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Sadanandan Velu

Professor

The University of Alabama

UNITED STATES

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Jung-Hyun Lee

Assistant Professor

Seoul National University

SOUTH KOREA

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Filomeen Haerynck

Ghent University

BELGIUM

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Kyle Buchan

The University of Texas Health Science Center at Houston

UNITED STATES

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Michiel van Gent

Research Interests

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Articles (13)

The dynamic interactions between virus infections and nonsense-mediated decay

Herpesviruses mimic zygotic genome activation to promote viral replication

Herpes simplex virus infection induces necroptosis of neurons and astrocytes in human fetal organotypic brain slice cultures

Neurovirulence of Usutu virus in human fetal organotypic brain slice cultures partially resembles Zika and West Nile virus.

Varicella-zoster virus proteome-wide T-cell screening demonstrates low prevalence of virus-specific CD8 T-cells in latently infected human trigeminal ganglia

GTF3A mutations predispose to herpes simplex encephalitis by disrupting biogenesis of the host-derived RIG-I ligand RNA5SP141

The US3 Kinase of Herpes Simplex Virus Phosphorylates the RNA Sensor RIG-I To Suppress Innate Immunity.

The herpesvirus accessory protein γ134.5 facilitates viral replication by disabling mitochondrial translocation of RIG-I

ISG15-dependent activation of the sensor MDA5 is antagonized by the SARS-CoV-2 papain-like protease to evade host innate immunity

Human-specific staphylococcal virulence factors enhance pathogenicity in a humanised zebrafish C5a receptor model

Nonsense-mediated decay controls the reactivation of the oncogenic herpesviruses EBV and KSHV

High-throughput RNA sequencing of paraformaldehyde-fixed single cells

Host–Receptor Post-Translational Modifications Refine Staphylococcal Leukocidin Cytotoxicity

Collaborators (10)

Jos A. G. Van Strijp

Nir Drayman

Tomasz Prajsnar

Michael Boettcher

Stephen A. Renshaw

Werner J.D. Ouwendijk

Sadanandan Velu

Jung-Hyun Lee

Filomeen Haerynck

Kyle Buchan

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