Petra Scholze

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Medical University of Vienna Center for Brain Research

Austria

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Recent Grants

Grant: Close

Human Nicotinic Receptor Polymorphisms in Synaptic Pathology

Open Date: 2018-04-01

Close Date: 2021-03-31

Grant: Close

Neuronal nicotinic acetylcholine receptors (nAChR) dissected

Open Date: 2007-01-01

Close Date: 2011-12-31

Grant: Close

Identification of Accessory Proteins Interact with Glycine Transporters

Open Date: 2004-02-01

Close Date: 2006-01-31

Articles (18)

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2024

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2024

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2024

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2023

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2023

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2022

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2022

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2022

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2022

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2022

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2021

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2020

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2020

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2020

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2020

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2020

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2020

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2020

Collaborators (13)

Margot Ernst

Associate Professor

Medical University of Vienna Center for Brain Research

AUSTRIA

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Abdeslam Chagraoui

Rouen University Hospital

FRANCE

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Filip Tadeusz Koniuszewski

Medical University of Vienna

AUSTRIA

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Florian Daniel Vogel

Universität Wien

AUSTRIA

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Martin Krenn

Medical University of Vienna

AUSTRIA

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Christina Birkedahl Falk-Petersen

Københavns Universitet

DENMARK

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Uwe MASKOS

Lab Chief

Institut Pasteur

FRANCE

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Wolfgang Kummer

GERMANY

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Matias Wagner

Technical University of Munich

GERMANY

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Silvia Madritsch

Medical University of Vienna

AUSTRIA

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Nils Ole Dalby

University of Copenhagen

DENMARK

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Elisabeth Graf

Klinikum rechts der Isar der Technischen Universität München

GERMANY

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Dominik S. Westphal

Technical University of Munich

GERMANY

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Petra Scholze

Research Interests

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Recent Grants

Articles (18)

The human‐specific nicotinic receptor subunit CHRFAM7A reduces α7 receptor function in human induced pluripotent stem cells‐derived and transgenic mouse neurons

Generation and Characterization of a Human Neuronal In Vitro Model for Rett Syndrome Using a Direct Reprogramming Method

Vascular effects of midazolam, flumazenil, and a novel imidazobenzodiazepine MP-III-058 on isolated rat aorta

Coronaridine congeners induce sedative and anxiolytic-like activity in naïve and stressed/anxious mice by allosteric mechanisms involving increased GABAA receptor affinity for GABA

Tricyclic antipsychotics and antidepressants can inhibit α5‐containing GABAA receptors by two distinct mechanisms

A de novo missense variant in GABRA4 alters receptor function in an epileptic and neurodevelopmental phenotype

Novel alpha6 preferring GABA-A receptor ligands based on loreclezole

Molecular Mingling: Multimodal Predictions of Ligand Promiscuity in Pentameric Ligand-Gated Ion Channels

GABA tonic currents and glial cells are altered during epileptogenesis in a mouse model of Dravet syndrome

A Benzodiazepine Ligand with Improved GABAA Receptor α5-Subunit Selectivity Driven by Interactions with Loop C

Allosteric Modulation of GABAA Receptors in Rat Basolateral Amygdala Blocks Stress-Enhanced Reacquisition of Nicotine Self-Administration

The α5 Nicotinic Acetylcholine Receptor Subunit Differentially Modulates α4β2<sup></sup> and α3β4<sup></sup> Receptors.

Nicotine stimulates ion transport via metabotropic β4 subunit containing nicotinic ACh receptors.

Two Distinct Populations of α1α6-Containing GABAA-Receptors in Rat Cerebellum.

Silencing of spontaneous activity at α4β1/3δ GABAA receptors in hippocampal granule cells reveals different ligand pharmacology

Coronaridine congeners potentiate GABAA receptors and induce sedative activity in mice in a benzodiazepine-insensitive manner

Acute nicotine administration stimulates ciliary activity via α3β4 nAChR in the mouse trachea.

Collaborators (13)

Margot Ernst

Abdeslam Chagraoui

Filip Tadeusz Koniuszewski

Florian Daniel Vogel

Martin Krenn

Christina Birkedahl Falk-Petersen

Uwe MASKOS

Wolfgang Kummer

Matias Wagner

Silvia Madritsch

Nils Ole Dalby

Elisabeth Graf

Dominik S. Westphal

Make your next move.

Petra Scholze

Research Interests

Explore related searches

Contact this professor

Recent Grants

Articles (18)

The human‐specific nicotinic receptor subunit CHRFAM7A reduces α7 receptor function in human induced pluripotent stem cells‐derived and transgenic mouse neurons

Generation and Characterization of a Human Neuronal In Vitro Model for Rett Syndrome Using a Direct Reprogramming Method

Vascular effects of midazolam, flumazenil, and a novel imidazobenzodiazepine MP-III-058 on isolated rat aorta

Coronaridine congeners induce sedative and anxiolytic-like activity in naïve and stressed/anxious mice by allosteric mechanisms involving increased GABAA receptor affinity for GABA

Tricyclic antipsychotics and antidepressants can inhibit α5‐containing GABAA receptors by two distinct mechanisms

A de novo missense variant in GABRA4 alters receptor function in an epileptic and neurodevelopmental phenotype

Novel alpha6 preferring GABA-A receptor ligands based on loreclezole

Molecular Mingling: Multimodal Predictions of Ligand Promiscuity in Pentameric Ligand-Gated Ion Channels

GABA tonic currents and glial cells are altered during epileptogenesis in a mouse model of Dravet syndrome

A Benzodiazepine Ligand with Improved GABAA Receptor α5-Subunit Selectivity Driven by Interactions with Loop C

Allosteric Modulation of GABAA Receptors in Rat Basolateral Amygdala Blocks Stress-Enhanced Reacquisition of Nicotine Self-Administration

The α5 Nicotinic Acetylcholine Receptor Subunit Differentially Modulates α4β2<sup>*</sup> and α3β4<sup>*</sup> Receptors.

Nicotine stimulates ion transport via metabotropic β4 subunit containing nicotinic ACh receptors.

Two Distinct Populations of α1α6-Containing GABAA-Receptors in Rat Cerebellum.

Silencing of spontaneous activity at α4β1/3δ GABAA receptors in hippocampal granule cells reveals different ligand pharmacology

Coronaridine congeners potentiate GABAA receptors and induce sedative activity in mice in a benzodiazepine-insensitive manner

Acute nicotine administration stimulates ciliary activity via α3β4 nAChR in the mouse trachea.

Collaborators (13)

Margot Ernst

Abdeslam Chagraoui

Filip Tadeusz Koniuszewski

Florian Daniel Vogel

Martin Krenn

Christina Birkedahl Falk-Petersen

Uwe MASKOS

Wolfgang Kummer

Matias Wagner

Silvia Madritsch

Nils Ole Dalby

Elisabeth Graf

Dominik S. Westphal

Make your next move.

The α5 Nicotinic Acetylcholine Receptor Subunit Differentially Modulates α4β2<sup></sup> and α3β4<sup></sup> Receptors.