Sachel Mok

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Assistant Professor of Medical Sciences

Columbia University Irving Medical Center

United States

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About

Dr. Sachel Mok is an Assistant Professor of Medical Sciences at Columbia University Irving Medical Center in the United States. His research focuses on the genetic and evolutionary aspects of malaria, particularly regarding multi-drug resistance mechanisms in Plasmodium falciparum . Recent publications include studies on the effects of recombination on drug resistance evolution and determinants of piperaquine-resistant malaria in South America.

Recent Grants

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Defining the genetic basis of piperaquine resistance in P. falciparum to support malaria elimination

Open Date: 2016-05-01

Close Date: 2019-04-30

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Collaborators (7)

Robert Zupko

Pennsylvania State University

UNITED STATES

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David Fidock

Professor

Columbia University

UNITED STATES

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Jerzy Michal Dziekan

Walter and Eliza Hall Institute of Medical Research

AUSTRALIA

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Philip Rosenthal

Professor

University of California, San Francisco

UNITED STATES

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Kely Rubiano

Research technician

Columbia University

UNITED STATES

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Gilles Courtemanche

Department head

BIOASTER

FRANCE

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Marcus Lee

Wellcome Sanger Institute

UNITED KINGDOM

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Sachel Mok

Research Interests

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About

Recent Grants

Articles (20)

Effects of recombination on multi-drug resistance evolution in Plasmodium falciparum malaria

Gametocyte production and transmission fitness of African and Asian <i>Plasmodium falciparum</i> isolates with differential susceptibility to artemisinins

Determinants of piperaquine-resistant malaria in South America

Generation of a mutator parasite to drive resistome discovery in Plasmodium falciparum

Potent acyl-CoA synthetase 10 inhibitors kill Plasmodium falciparum by disrupting triglyceride formation

Determinants of piperaquine-resistant malaria in South America.

Generation of a mutator parasite to drive resistome discovery in <i>Plasmodium falciparum</i>

Mapping the genomic landscape of multidrug resistance in <i>Plasmodium falciparum</i> and its impact on parasite fitness

Mapping the genomic landscape of multidrug resistance in Plasmodium falciparum and its impact on parasite fitness

Potent acyl-CoA synthetase 10 inhibitors kill <i>Plasmodium falciparum</i> by disrupting triglyceride formation

Short tandem repeat polymorphism in the promoter region of cyclophilin 19B drives its transcriptional upregulation and contributes to drug resistance in the malaria parasite <i>Plasmodium falciparum</i>

Short tandem repeat polymorphism in the promoter region of cyclophilin 19B drives its transcriptional upregulation and contributes to drug resistance in the malaria parasite Plasmodium falciparum

Comparative Analysis of Plasmodium falciparum Genotyping via SNP Detection, Microsatellite Profiling, and Whole-Genome Sequencing

The antimalarial MMV688533 provides potential for single-dose cures with a high barrier to Plasmodium falciparum parasite resistance

Plasmodium falciparum K13 mutations in Africa and Asia impact artemisinin resistance and parasite fitness

3-Hydroxy-propanamidines, a New Class of Orally Active Antimalarials Targeting Plasmodium falciparum

Artemisinin-resistant K13 mutations rewire Plasmodium falciparum’s intra-erythrocytic metabolic program to enhance survival

Molecular Mechanisms of Drug Resistance in Plasmodium falciparum Malaria

Inhibition of Resistance-Refractory P. falciparum Kinase PKG Delivers Prophylactic, Blood Stage, and Transmission-Blocking Antiplasmodial Activity

Local emergence in Amazonia of Plasmodium falciparum k13 C580Y mutants associated with in vitro artemisinin resistance

Collaborators (7)

Robert Zupko

David Fidock

Jerzy Michal Dziekan

Philip Rosenthal

Kely Rubiano

Gilles Courtemanche

Marcus Lee

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