Sandra Galic

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St Vincent's Institute of Medical Research

Australia

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Control of catabolic and anabolic pathways by AMPK

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Control of appetite by AMPK

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Inhibition of AMPK signalling as a strategy for decreasing appetite

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The Role of AMPK in Suppressing Liver Steatosis and Atherosclerosis

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Articles (12)

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2025

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2024

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2022

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2022

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2022

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2022

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2021

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2021

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2021

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2020

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2020

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2020

Collaborators (11)

Janni Petersen

Flinders University

AUSTRALIA

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Itamar Levinger

AUSTRALIA

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Thomas Loudovaris

Research Professor

University of Arizona

UNITED STATES

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Kim Loh

St Vincent's Institute of Medical Research

AUSTRALIA

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John Scott

Lab Head, Neurometabolism

Monash Institute of Pharmaceutical Sciences

AUSTRALIA

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Alba Moreno‐Asso

AUSTRALIA

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Kei Sakamoto

Professor

University of Copenhagen

DENMARK

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David Ross Laybutt

Westmead Institute for Medical Research

AUSTRALIA

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Nolan Hoffman

Senior Lecturer

Australian Catholic University

AUSTRALIA

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Andrew L. Gundlach

AUSTRALIA

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Jon Oakhill

Australian Catholic University

AUSTRALIA

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Sandra Galic

Research Interests

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Recent Grants

Articles (12)

Metformin suppresses atherosclerosis by dampening extramedullary myelopoiesis

New developments in AMPK and mTORC1 cross-talk

Blocking AMPK β1 myristoylation enhances AMPK activity and protects mice from high-fat diet-induced obesity and hepatic steatosis

An AMPKα2-specific phospho-switch controls lysosomal targeting for activation

Defective AMPK regulation of cholesterol metabolism accelerates atherosclerosis by promoting HSPC mobilization and myelopoiesis

Structure-function analysis of the AMPK activator SC4 and identification of a potent pan AMPK activator

Neuropeptide Y1 receptor antagonism protects β-cells and improves glycemic control in type 2 diabetes.

Regulation of Pancreatic β-Cell Function by the NPY System

Molecular mechanisms underlying the beneficial effects of exercise on brain function and neurological disorders

CaMKK2 is inactivated by cAMP-PKA signaling and 14-3-3 adaptor proteins

Functional analysis of an R311C variant of Ca<sup>2+</sup>-calmodulin-dependent protein kinase kinase-2 (CaMKK2) found as a de novo mutation in a patient with bipolar disorder

CaMKK2 is inactivated by cAMP-PKA signaling and 14-3-3 adaptor proteins.

Collaborators (11)

Janni Petersen

Itamar Levinger

Thomas Loudovaris

Kim Loh

John Scott

Alba Moreno‐Asso

Kei Sakamoto

David Ross Laybutt

Nolan Hoffman

Andrew L. Gundlach

Jon Oakhill

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