Steven Bergink

assistant professor/senior lecturer

Netherlands

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Dr. Steven Bergink is an Assistant Professor and Senior Lecturer at University College Groningen in the Netherlands. His research focuses on the interplay between genomic instability and protein homeostasis, with recent articles exploring topics such as glioblastoma, DNAJB chaperones, and the effects of oxidative stress on cellular integrity. He is actively involved in advancing the understanding of protein aggregation-related diseases, contributing to fields including cellular stress responses and molecular biology.

Articles (12)

Year:

2024

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2022

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2022

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2022

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2021

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2021

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2021

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2021

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2021

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2020

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2020

Year:

2020

Collaborators (13)

Liesbeth M. Veenhoff

NETHERLANDS

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Ben N. G. Giepmans

NETHERLANDS

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Muriel Mari

Aarhus University

DENMARK

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Harm Kampinga

Full Professor, Head of Department

University Medical Center Groningen

NETHERLANDS

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Heath Ecroyd

Professor

University of Wollongong

AUSTRALIA

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Veena Prahlad

Iowa State University

UNITED STATES

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Emma Gerrits

Karolinska Institutet

SWEDEN

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Bart J. L. Eggen

NETHERLANDS

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Andrew Truman

Professor & Associate Chair of Research

University of North Carolina at Charlotte

UNITED STATES

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Nadinath Nillegoda

MONASH UNIVERSITY

AUSTRALIA

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Sybille Krauss

Professor

University of Siegen

GERMANY

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Robert Henning

Professor of Pharmacology

University Medical Center Groningen

NETHERLANDS

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Olivier Genest

FRANCE

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Steven Bergink

Research Interests

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About

Articles (12)

Glioblastoma and its treatment are associated with extensive accelerated brain aging

Cooling of Cells and Organs Confers Extensive DNA Strand Breaks Through Oxidative Stress and ATP Depletion

The chaperone DNAJB6 surveils FG-nucleoporins and is required for interphase nuclear pore complex biogenesis

Targeting DNA topoisomerases or checkpoint kinases results in an overload of chaperone systems, triggering aggregation of a metastable subproteome

DNAJB chaperones suppress destabilised protein aggregation via a region distinct from that used to inhibit amyloidogenesis

First Virtual International Congress on Cellular and Organismal Stress Responses, November 5-6, 2020.

FOXO1 controls protein synthesis and transcript abundance of mutant polyglutamine proteins, preventing protein aggregation

Genome instability and loss of protein homeostasis: converging paths to neurodegeneration?

Locked in a vicious cycle: the connection between genomic instability and a loss of protein homeostasis

Functional diversity between HSP70 paralogs caused by variable interactions with specific co-chaperones.

DNAJB6, a Key Factor in Neuronal Sensitivity to Amyloidogenesis

Insulin-like growth factor 2 (IGF2) protects against Huntington's disease through the extracellular disposal of protein aggregates.

Collaborators (13)

Liesbeth M. Veenhoff

Ben N. G. Giepmans

Muriel Mari

Harm Kampinga

Heath Ecroyd

Veena Prahlad

Emma Gerrits

Bart J. L. Eggen

Andrew Truman

Nadinath Nillegoda

Sybille Krauss

Robert Henning

Olivier Genest

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