Takahiro Seki

Visiting Professor

Kumamoto University

Japan

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About

Takahiro Seki is a Visiting Professor at Kumamoto University in Japan. His research focuses on neurological dysfunction, particularly in relation to intracerebral hemorrhage, and involves exploring mechanisms such as SUMOylation and pharmacological interventions. Recent studies include the investigation of Nurr1 overexpression and the effects of novel compounds on autophagy and inflammatory responses. Professor Seki contributes significantly to understanding cerebral blood flow regulation and treatment approaches for neurological disorders.

Articles (21)

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2021

Collaborators (14)

Yuki Kondo

Associate Professor

Kumamoto University

JAPAN

JAPAN

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Hirokazu Hirai

Professor & Chair

Gunma University Graduate School of Medicine

JAPAN

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Associate Professor

Kumamoto University

JAPAN

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Takahiro Seki

Research Interests

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About

Articles (21)

Inhibition of SUMOylation exacerbates neurological dysfunction and delays hematoma clearance after intracerebral hemorrhage in mice

Menaquinone-4 Alleviates Neurological Deficits Associated with Intracerebral Hemorrhage by Preserving Corticospinal Tract in Mice

Identification of a novel aromatic-turmerone analog that activates chaperone-mediated autophagy through the persistent activation of p38

Nurr1 overexpression in the primary motor cortex alleviates motor dysfunction induced by intracerebral hemorrhage in the striatum in mice

Goreisan regulates cerebral blood flow according to barometric pressure fluctuations in female C57BL/6J mice

Intracerebroventricular 2-hydroxypropyl-γ-cyclodextrin alleviates hepatic manifestations without distributing to the liver in a murine model of Niemann-Pick disease type C

Structurally Distinct Nurr1 Ligands Exhibit Different Pharmacological Characteristics in Regulating Inflammatory Responses of Microglial BV-2 Cells

Wild-type and pathogenic forms of ubiquilin 2 differentially modulate components of the autophagy-lysosome pathways

Acyclic retinoid peretinoin reduces hemorrhage-associated brain injury in vitro and in vivo

Matcha Tea Powder’s Antidepressant-like Effect through the Activation of the Dopaminergic System in Mice Is Dependent on Social Isolation Stress

Therapeutic effect of allicin in a mouse model of intracerebral hemorrhage

Different solubilizing ability of cyclodextrin derivatives for cholesterol in Niemann–Pick disease type C treatment

Wild-type and pathogenic forms of ubiquilin 2 differentially modulate components of the autophagy-lysosome pathways

D-Cysteine Activates Chaperone-Mediated Autophagy in Cerebellar Purkinje Cells via the Generation of Hydrogen Sulfide and Nrf2 Activation

Fine-tuned cholesterol solubilizer, mono-6-O-α-D-maltosyl-γ-cyclodextrin, ameliorates experimental Niemann–Pick disease type C without hearing loss

A Nurr1 ligand C-DIM12 attenuates brain inflammation and improves functional recovery after intracerebral hemorrhage in mice

Distinct Pharmacological Profiles of Monosulfide and Trisulfide in an Experimental Model of Intracerebral Hemorrhage in Mice

Hydroxychloroquine improves motor function and affords neuroprotection without inhibition of inflammation and autophagy in mice after intracerebral hemorrhage

Cell-penetrating albumin enhances the sublingual delivery of antigens through macropinocytosis

Differential mode of cholesterol inclusion with 2‐hydroxypropyl‐cyclodextrins increases safety margin in treatment of Niemann‐Pick disease type C

Therapeutic potential of d-cysteine against in vitro and in vivo models of spinocerebellar ataxia

Collaborators (14)

Yuki Kondo

Kentaro Minami

Toru Miwa

Hiroshi Katsuki

Alex Boateng

Tetsumi Irie

Natsuko Hitora-Imamura

Naoki Yoshikawa

Hirokazu Hirai

Lisa Sharkey

Toru Takeo

Ayumu Konno

Yara Atef Ahmed

Tsuyoshi Shuto

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