Tatsuya Morishima

Lecturer

Kumamoto University

Japan

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About

Tatsuya Morishima is a Lecturer at Kumamoto University in Japan. His research focuses on hematopoietic differentiation, leukemia mechanisms, and the role of mitochondria in cell metabolism. Recent publications include studies on erythroid differentiation, hematopoietic stem cell expansion, and the metabolic adaptations in leukemia cells.

Articles (15)

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2025

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2024

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2024

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2023

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2023

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2023

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2023

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2023

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2022

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2022

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2022

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2021

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2021

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2021

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2020

Collaborators (13)

Takeshi Masuda

Kumamoto University

JAPAN

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Hitoshi Takizawa

Vice Director and Professor

Kumamoto University

JAPAN

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Fabienne Meier-Abt

University Hospital of Zurich

SWITZERLAND

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Ken Ishii

Professor

University of Tokyo

JAPAN

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Julia Skokowa

Professor and Head of Division of Translational Oncology

University Hospital Tübingen

GERMANY

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Takaaki Akaike

Tohoku University graduate school of medicine

JAPAN

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Fan-Yan Wei

Professor

Tohoku University

JAPAN

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Masayuki Kai

Vice President, Head of Research

UNITED STATES

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Yoshikazu Hayashi

Assistant professor

Fukuoka Dental College

JAPAN

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Shinji Fukuda

Keio University

JAPAN

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Yuichiro Arima

Kumamoto University

JAPAN

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Guy Sauvageau

Affiliated professor

Université de Montréal

CANADA

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Masao Matsuoka

Kumamoto University

JAPAN

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Tatsuya Morishima

Research Interests

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Articles (15)

Mitochondrial translation regulates terminal erythroid differentiation by maintaining iron homeostasis

Expansion effect of romiplostim on hematopoietic stem and progenitor cells versus thrombopoietin and eltrombopag.

Phospholipid metabolic adaptation promotes survival of IDH2 mutant acute myeloid leukemia cells

Humanized mouse models with endogenously developed human natural killer cells for in vivo immunogenicity testing of HLA class I-edited iPSC-derived cells

Correction: NAMPT/SIRT2-mediated inhibition of the p53-p21 signaling pathway is indispensable for maintenance and hematopoietic differentiation of human iPS cells

GPRC5C drives branched-chain amino acid metabolism in leukemogenesis

Akkermansia muciniphila induces slow extramedullary hematopoiesis via cooperative IL‐1R/TLR signals

Erratum: iPSC modeling of stage-specific leukemogenesis reveals BAALC as a key oncogene in severe congenital neutropenia (Cell Stem Cell (2021) 28(5) (906–922.e6), (S1934590921001569), (10.1016/j.stem.2021.03.023))

Hematopoietic stem and progenitor cells integrate microbial signals to promote post‐inflammation gut tissue repair

Unique molecular and functional features of extramedullary hematopoietic stem and progenitor cell reservoirs in humans

iPSC modeling of stage-specific leukemogenesis reveals BAALC as a key oncogene in severe congenital neutropenia.

Fit-For-All iPSC-Derived Cell Therapies and Their Evaluation in Humanized Mice With NK Cell Immunity.

NAMPT/SIRT2-mediated inhibition of the p53-p21 signaling pathway is indispensable for maintenance and hematopoietic differentiation of human iPS cells.

Genetic fingerprint defines hematopoietic stem cell pool size and function.

Collaborators (13)

Takeshi Masuda

Hitoshi Takizawa

Fabienne Meier-Abt

Ken Ishii

Julia Skokowa

Takaaki Akaike

Fan-Yan Wei

Masayuki Kai

Yoshikazu Hayashi

Shinji Fukuda

Yuichiro Arima

Guy Sauvageau

Masao Matsuoka

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