Wei Gu

Professor

Columbia University

United States

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About

Professor Wei Gu is a faculty member at Columbia University in the United States. His research focuses on the regulation of cellular pathways related to p53, ferroptosis, and tumorigenesis. Recent articles by Professor Gu examine complex interactions in transcriptional activation, the implications of non-apoptotic cell death in cancer, and strategies for targeting tumor growth through various molecular mechanisms.

Articles (18)

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2025

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2025

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2025

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2024

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2024

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2024

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2024

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2024

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2022

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2022

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2022

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2022

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2021

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2021

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2021

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2020

Collaborators (4)

Brent Stockwell

Professor and Chair

Columbia University

UNITED STATES

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Zhiguo Zhang

Professor

Columbia University Irving Medical Center

UNITED STATES

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Holger Gerhardt

Group Leader/Professor

GERMANY

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Yan Mo

NYU Langone Medical Center

UNITED STATES

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Wei Gu

Research Interests

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About

Articles (18)

p53-regulated non-apoptotic cell death pathways and their relevance in cancer and other diseases

Protein kinase A regulates ferroptosis by controlling GPX4 m<sup>6</sup>A modification through phosphorylation of ALKBH5.

The PURB–HOTAIR complex regulates p53-dependent promoter-specific transcriptional activation

GAS41 modulates ferroptosis by anchoring NRF2 on chromatin

An unexpected role for the ketogenic diet in triggering tumor metastasis by modulating BACH1-mediated transcription

Understanding the complexity of p53 in a new era of tumor suppression

Regulation of SLC7A11 as an unconventional checkpoint in tumorigenesis through ferroptosis

Ferroptosis as a new tool for tumor suppression through lipid peroxidation

Inhibition of USP7 induces p53-independent tumor growth suppression in triple-negative breast cancers by destabilizing FOXM1

Targeting USP2 regulation of VPRBP-mediated degradation of p53 and PD-L1 for cancer therapy

Epigenome Programming by H3.3K27M Mutation Creates a Dependence of Pediatric Glioma on SMARCA4

The complexity of p53-mediated metabolic regulation in tumor suppression

Deciphering the acetylation code of p53 in transcription regulation and tumor suppression

p53 in ferroptosis regulation: the new weapon for the old guardian

An unexpected role for Dicer as a reader of the unacetylated DNA binding domain of p53 in transcriptional regulation

iPLA2β-mediated lipid detoxification controls p53-driven ferroptosis independent of GPX4

The deubiquitinase USP11 regulates cell proliferation and ferroptotic cell death via stabilization of NRF2 USP11 deubiquitinates and stabilizes NRF2

SIRT7 activates p53 by enhancing PCAF-mediated MDM2 degradation to arrest the cell cycle

Collaborators (4)

Brent Stockwell

Zhiguo Zhang

Holger Gerhardt

Yan Mo

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