Wei Shi

Professor

MONASH UNIVERSITY

Australia

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About

Wei Shi is a Professor at Monash University, Australia. His research primarily focuses on tumor immunity, cellular crosstalk in the tumor microenvironment, and the mechanisms of immune regulation. Recent publications include investigations into transcript expression analysis, the role of cytokines in T cell responses, and innovative methodologies for single-cell RNA sequencing data analysis.

Articles (10)

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2024

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2024

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2024

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2024

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2023

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2023

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2023

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2022

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2021

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2021

Collaborators (13)

Matthew Ritchie

Laboratory Head

Walter and Eliza Hall Institute of Medical Research

AUSTRALIA

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Gunther Doehlemann

GERMANY

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Isaak Quast

MONASH UNIVERSITY

AUSTRALIA

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Zhoujie Ding

MONASH UNIVERSITY

AUSTRALIA

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Yannick O Alexandre

AUSTRALIA

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Andrew Douglas Lucas

University of Western Australia

AUSTRALIA

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William Heath

Professor

University of Melbourne

AUSTRALIA

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John Mariadason

La Trobe University

AUSTRALIA

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Gordon Smyth

Honorary Professorial Fellow

University of Melbourne

AUSTRALIA

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Marcus Robinson

MONASH UNIVERSITY

AUSTRALIA

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Maximilien Evrard

University of Melbourne

AUSTRALIA

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Lisa A. Mielke

AUSTRALIA

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Ryan O'Keefe

Olivia Newton-John Cancer Research Institute

AUSTRALIA

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Wei Shi

Research Interests

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Articles (10)

Mechanisms of cellular crosstalk in the gastric tumor microenvironment are mediated by YAP1 and STAT3

Dividing out quantification uncertainty allows efficient assessment of differential transcript expression with edgeR.

Author Correction: CIS is a potent checkpoint in NK cell–mediated tumor immunity (Nature Immunology, (2016), 17, 7, (816-824), 10.1038/ni.3470)

Author Correction: c-Maf-dependent Treg cell control of intestinal TH17 cells and IgA establishes host–microbiota homeostasis (Nature Immunology, (2019), 20, 4, (471-481), 10.1038/s41590-019-0316-2)

Combination of in vivo proximity labeling and co-immunoprecipitation identifies the host target network of a tumor-inducing effector in the fungal maize pathogen Ustilago maydis

cellCounts: an R function for quantifying 10x Chromium single-cell RNA sequencing data.

A tuft cell - ILC2 signaling circuit provides therapeutic targets to inhibit gastric metaplasia and tumor development

CD137L and CD4 T cells limit BCL6‐expressing pre‐germinal center B cell expansion and BCL6‐driven B cell malignancy

Discrete tissue microenvironments instruct diversity in resident memory T cell function and plasticity

Host il11 signaling suppresses cd4+ t cell-mediated antitumor responses to colon cancer in mice

Collaborators (13)

Matthew Ritchie

Gunther Doehlemann

Isaak Quast

Zhoujie Ding

Yannick O Alexandre

Andrew Douglas Lucas

William Heath

John Mariadason

Gordon Smyth

Marcus Robinson

Maximilien Evrard

Lisa A. Mielke

Ryan O'Keefe

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