Alessandro Sartori

University of Zurich

Switzerland

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Articles (14)

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2025

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2024

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2023

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2022

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2021

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2021

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2021

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2021

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2021

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2021

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2021

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2021

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2021

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2020

Collaborators (13)

Joanna I Loizou

AUSTRIA

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Peter K Vogt

Professor

Scripps Research Institute

UNITED STATES

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Matthias Altmeyer

University of Zurich

SWITZERLAND

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Claus Sorensen

Københavns Universitet

DENMARK

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Nicholas Allen

Professor

Cardiff University

UNITED KINGDOM

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Sven Rottenberg

University of Bern

SWITZERLAND

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Sarah Aldous

University College London

UNITED KINGDOM

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Serena Nik-Zainal

NIHR Research Professor & Honorary Consultant

University of Cambridge

UNITED KINGDOM

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Masataka Tsuda

Chief

National Institute of Health Sciences

JAPAN

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Joseph Hamilton

University College London

UNITED KINGDOM

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Mark Dillingham

University of Bristol

UNITED KINGDOM

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Virginie Ropars

Researcher

CEA Saclay

FRANCE

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Pablo Huertas

Universidad de Sevilla

SPAIN

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Alessandro Sartori

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Articles (14)

Cancer-cell-derived cGAMP limits the activity of tumor-associated CD8<sup>+</sup> T cells.

H2AX promotes replication fork degradation and chemosensitivity in BRCA-deficient tumours

PLK1 regulates CtIP and DNA2 interplay in long-range DNA end resection

RNAi Screening Uncovers a Synthetic Sick Interaction between CtIP and the BARD1 Tumor Suppressor.

A stapled peptide mimetic of the CtIP tetramerization motif interferes with double-strand break repair and replication fork protection

FAN1 controls mismatch repair complex assembly via MLH1 retention to stabilize CAG repeat expansion in Huntington's disease.

FANCD2-Associated Nuclease 1 Partially Compensates for the Lack of Exonuclease 1 in Mismatch Repair.

FAN1-MLH1 interaction affects repair of DNA interstrand cross-links and slipped-CAG/CTG repeats

Human CtIP: A ‘double agent’ in DNA repair and tumorigenesis

Stereo- and regiodefined DNA-encoded chemical libraries enable efficient tumour-targeting applications.

FAN1, a DNA Repair Nuclease, as a Modifier of Repeat Expansion Disorders

FAN1, a DNA Repair Nuclease, as a Modifier of Repeat Expansion Disorders

A Single‐Stranded DNA‐Encoded Chemical Library Based on a Stereoisomeric Scaffold Enables Ligand Discovery by Modular Assembly of Building Blocks

Collaborators (13)

Joanna I Loizou

Peter K Vogt

Matthias Altmeyer

Claus Sorensen

Nicholas Allen

Sven Rottenberg

Sarah Aldous

Serena Nik-Zainal

Masataka Tsuda

Joseph Hamilton

Mark Dillingham

Virginie Ropars

Pablo Huertas

Make your next move.

Alessandro Sartori

Research Interests

Explore related searches

Contact this professor

Articles (14)

Cancer-cell-derived cGAMP limits the activity of tumor-associated CD8&lt;sup&gt;+&lt;/sup&gt; T cells.

H2AX promotes replication fork degradation and chemosensitivity in BRCA-deficient tumours

PLK1 regulates CtIP and DNA2 interplay in long-range DNA end resection

RNAi Screening Uncovers a Synthetic Sick Interaction between CtIP and the BARD1 Tumor Suppressor.

A stapled peptide mimetic of the CtIP tetramerization motif interferes with double-strand break repair and replication fork protection

FAN1 controls mismatch repair complex assembly via MLH1 retention to stabilize CAG repeat expansion in Huntington's disease.

FANCD2-Associated Nuclease 1 Partially Compensates for the Lack of Exonuclease 1 in Mismatch Repair.

FAN1-MLH1 interaction affects repair of DNA interstrand cross-links and slipped-CAG/CTG repeats

Human CtIP: A ‘double agent’ in DNA repair and tumorigenesis

Stereo- and regiodefined DNA-encoded chemical libraries enable efficient tumour-targeting applications.

FAN1, a DNA Repair Nuclease, as a Modifier of Repeat Expansion Disorders

FAN1, a DNA Repair Nuclease, as a Modifier of Repeat Expansion Disorders

A Single‐Stranded DNA‐Encoded Chemical Library Based on a Stereoisomeric Scaffold Enables Ligand Discovery by Modular Assembly of Building Blocks

Collaborators (13)

Joanna I Loizou

Peter K Vogt

Matthias Altmeyer

Claus Sorensen

Nicholas Allen

Sven Rottenberg

Sarah Aldous

Serena Nik-Zainal

Masataka Tsuda

Joseph Hamilton

Mark Dillingham

Virginie Ropars

Pablo Huertas

Make your next move.

Cancer-cell-derived cGAMP limits the activity of tumor-associated CD8<sup>+</sup> T cells.