Hannes Lans

Associate Professor

Erasmus MC

Netherlands

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About

Dr. Hannes Lans is an Associate Professor at Erasmus MC in the Netherlands. His research focuses on the mechanisms of DNA repair, particularly in relation to transcription-coupled repair and responses to DNA lesions. Through his recent publications, he has explored topics such as the dynamics of RNA polymerase II at DNA damage sites and the role of ubiquitin ligases in repair processes. His work significantly contributes to the understanding of cellular responses to genomic instability and associated diseases.

Articles (18)

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2024

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2022

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2021

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2020

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2020

Collaborators (8)

Jeroen Demmers

Associate Professor | Director of the Proteomics Core Facility

Erasmus MC

NETHERLANDS

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Ryan Barnes

Assistant Professor

University of Kansas Medical Center

UNITED STATES

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NITIKA TANEJA

Associate Professor

Erasmus MC

NETHERLANDS

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Jurgen Marteijn

Full Professor

Erasmus MC

NETHERLANDS

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Petra Heffeter

Full professor

Medical University of Vienna

AUSTRIA

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Titia K Sixma

NETHERLANDS

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Amandine Moretton

Université Clermont Auvergne

FRANCE

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Wim Vermeulen

Professor Genetic Stability

Erasmus University Medical Center

NETHERLANDS

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Hannes Lans

Research Interests

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Articles (18)

Differential processing of RNA polymerase II at DNA damage correlates with transcription-coupled repair syndrome severity

The small CRL4<sup>CSA</sup> ubiquitin ligase component DDA1 regulates transcription-coupled repair dynamics.

Persistent TFIIH binding to non-excised DNA damage causes cell and developmental failure.

Transcription-coupled DNA-protein crosslink repair by CSB and CRL4<sup>CSA</sup>-mediated degradation.

Author Correction: R-loop-derived cytoplasmic RNA–DNA hybrids activate an immune response

Recovery of protein synthesis to assay DNA repair activity in transcribed genes in living cells and tissues

R-loop-derived cytoplasmic RNA–DNA hybrids activate an immune response

Detection of oxaliplatin- and cisplatin-DNA lesions requires different global genome repair mechanisms that affect their clinical efficacy.

Trichothiodystrophy-associated MPLKIP maintains DBR1 levels for proper lariat debranching and ectodermal differentiation.

TFIIH central activity in nucleotide excision repair to prevent disease.

Global and transcription-coupled repair of 8-oxoG is initiated by nucleotide excision repair proteins

XPG: a multitasking genome caretaker

SMARCAD1-mediated active replication fork stability maintains genome integrity

C. elegans TFIIH subunit GTF-2H5/TTDA is a non-essential transcription factor indispensable for DNA repair

C. elegans survival assays to discern global and transcription-coupled nucleotide excision repair

Tissue-Specific DNA Repair Activity of ERCC-1/XPF-1

Ubiquitin and TFIIH-stimulated DDB2 dissociation drives DNA damage handover in nucleotide excision repair

ERCC1–XPF targeting to psoralen–DNA crosslinks depends on XPA and FANCD2

Collaborators (8)

Jeroen Demmers

Ryan Barnes

NITIKA TANEJA

Jurgen Marteijn

Petra Heffeter

Titia K Sixma

Amandine Moretton

Wim Vermeulen

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