Joerg Buescher

Head of Metabolomics Core Facility

Max Planck Institute of Immunobiology and Epigenetics

Germany

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About

Joerg Buescher is the Head of the Metabolomics Core Facility at the Max-Planck-Institut für Immunbiologie und Epigenetik in Germany. His research focuses on the metabolic aspects of immunology and neurobiology, as evidenced by his recent work on gut microbiota-derived metabolites and their effects on CNS-GVHD, as well as the interplay between microglia and neurons. Additionally, he investigates the role of metabolism in hematology and the critical functions of peroxisomes in immune cell survival.

Articles (20)

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Collaborators (22)

Philipp Demling

RWTH Aachen University

GERMANY

Joerg Buescher

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Articles (20)

Gut microbiota-derived TMAVA is a modulator of acute CNS-GVHD.

Acute suppression of mitochondrial ATP production prevents apoptosis and provides an essential signal for NLRP3 inflammasome activation

Microglia–neuron crosstalk through Hex–GM2–MGL2 maintains brain homeostasis

Differentiation, ageing and leukaemia alter the metabolic profile of human bone marrow haematopoietic stem and progenitor cells

Metabolism in hematology: Technological advances open new perspectives on disease biology and treatment

Peroxisomes are critical for a unique metabolic demand and survival of alveolar macrophages

Acetol biosynthesis enables NADPH balance during nitrogen limitation in engineered Escherichia coli

Neutrophil trapping and nexocytosis, mast cell-mediated processes for inflammatory signal relay

Rapid phagosome isolation enables unbiased multiomic analysis of human microglial phagosomes

Oncogene-induced TIM-3 ligand expression dictates susceptibility to anti–TIM-3 therapy in mice

Repair of airway epithelia requires metabolic rewiring towards fatty acid oxidation

Metabolic rewiring tunes dermal macrophages in staphylococcal skin infection

Phosphoinositide acyl chain saturation drives CD8+ effector T cell signaling and function

Editorial: Methods in Metabolomics 2022

LC-MS-Based Targeted Metabolomics for FACS-Purified Rare Cells

An LKB1-mitochondria axis controls T<sub>H</sub>17 effector function.

TFEB induces mitochondrial itaconate synthesis to suppress bacterial growth in macrophages.

Single-cell profiling of vascular endothelial cells reveals progressive organ-specific vulnerabilities during obesity.

Targeting MDM2 enhances antileukemia immunity after allogeneic transplantation via MHC-II and TRAIL-R1/2 upregulation

Targeted LC-MS/MS-based metabolomics and lipidomics on limited hematopoietic stem cell numbers

Collaborators (22)

Philipp Demling

Stephanie Christine Ganal-Vonarburg

Andrew Khalil

Katrin Kierdorf

Julio Saez-Rodriguez

james vince

Julia Kolter

Philipp Henneke

Natalie Köhler

Peter van Galen

Peter Carmeliet

Andreas Wack

Petya Apostolova

Basak Corak

Stefania Crotta

Matteo Villa

Oliver Gorka

Ramon Klein Geltink

Nina Cabezas Wallscheid

David E Sanin

Nora Klöting

Eirini Trompouki

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