Roger Preston

Lecturer in Vascular Biology

Ireland

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About

Roger Preston is a Lecturer in Vascular Biology at the Royal College of Surgeons in Ireland, specifically within the Department of Molecular and Cellular Therapeutics. His research largely focuses on thrombogenicity, myeloid cell behavior in thromboinflammatory diseases, and the role of inflammation in cardiometabolic diseases. Recent publications include investigations into the clinical phenotype of low VWF, the mechanisms of myeloid cell hypercoagulability, and the impact of environmental factors on health outcomes.

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2022

Collaborators (18)

Emily Day

Assistant Professor

University of Western Ontario

CANADA

Roger Preston

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Articles (25)

Clinical phenotype and pathophysiological mechanisms underlying qualitative Low VWF.

Trained Immunity Causes Myeloid Cell Hypercoagulability

Enhanced α2-3 linked sialylation determines the extended half-life of CHO-rVWF.

Tissue factor-dependent colitogenic CD4+ T cell thrombogenicity is regulated by activated protein C signalling

Sepsis-induced NET formation requires MYD88 but is independent of GSDMD and PAD4.

Type 1 VWD classification revisited: novel insights from combined analysis of the LoVIC and WiN studies

Mimicking activated protein C-progress by PARtnering peptides

Aptamer BT200 blocks interaction of K1405-1408 in the VWF-A1 domain with macrophage LRP1

Peptidylarginine deiminase 4: casting the NET over obesity?

The Role of Myeloid Cells in Thromboinflammatory Disease.

Endothelial cell activation, Weibel-Palade body secretion, and enhanced angiogenesis in severe COVID-19

Heterogeneity in the half-life of factor VIII concentrate in patients with hemophilia A is due to variability in the clearance of endogenous von Willebrand factor

Targeting NETs using dual-active DNase1 variants.

Dimethyl fumarate and 4-octyl itaconate are anticoagulants that suppress Tissue Factor in macrophages via inhibition of Type I Interferon.

Unraveling coagulation factor-mediated cellular signaling.

Von Willebrand factor-inflammation crosstalk in deep vein thrombosis.

Macrophage Galactose Lectin Contributes to the Regulation of FVIII (Factor VIII) Clearance in Mice-Brief Report.

VWF-ADAMTS13 axis dysfunction in children with sickle cell disease treated with hydroxycarbamide vs blood transfusion.

Publisher Correction: Dimethyl fumarate and 4-octyl itaconate are anticoagulants that suppress Tissue Factor in macrophages via inhibition of Type I Interferon.

Inflammation and cardiometabolic diseases induced by persistent organic pollutants and nutritional interventions: Effects of multi-organ interactions

Glycolytic reprogramming fuels myeloid cell-driven hypercoagulability.

New targeted therapy for hemophilia

Dysregulated haemostasis in thrombo-inflammatory disease.

The circadian clock influences T cell responses to vaccination by regulating dendritic cell antigen processing.

Intrinsic coagulation pathway-mediated thrombin generation in mouse whole blood.

Collaborators (18)

Emily Day

Edmund Gilbert

Siobhan Glavey

judith coppinger

Megan Kennedy

Luke O'Neill

James O'Donnell

Emma Creagh

Jacob Odeberg

Padraic Fallon

Mieszko Wilk

Jonas Emsley

Frank Leebeek

Lynn M. Butler

Johanna G. van der Bom

Hauke Weiss

Michael Carty

Gavin McManus

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