Werner J.H. Koopman

Associate professor

Radboud University Medical Center

Netherlands

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Werner J.H. Koopman is an Associate Professor at Radboud University Nijmegen Medical Centre, Netherlands. His research primarily focuses on mitochondrial function and its implications in various diseases, including cancer and neurodegenerative disorders. Recent articles by Dr. Koopman delve into mitochondrial profiling, energy metabolism, and the role of autophagy in ferroptosis, highlighting his contributions to understanding cellular metabolism and responses in pathological conditions.

Articles (19)

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2025

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2022

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2021

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2021

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2021

Collaborators (18)

Niamh Connolly

Lecturer

Royal College of Surgeons in Ireland

IRELAND

Werner J.H. Koopman

Research Interests

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Articles (19)

Mitochondrial Morphofunctional Profiling in Primary Human Skin Fibroblasts Using TMRM and Mitotracker Green Co-staining.

International consensus guidelines for the definition, detection, and interpretation of autophagy-dependent ferroptosis.

Metabolic alterations in fibroblasts of patients presenting with the MPAN subtype of neurodegeneration with brain iron accumulation (NBIA).

Ndufs4 knockout mice with isolated complex I deficiency engage a futile adaptive brain response.

Mitochondrial Nicotinamide Nucleotide Transhydrogenase: Role in Energy Metabolism, Redox Homeostasis, and Cancer.

Metabolic impact of genetic and chemical ADP/ATP carrier inhibition in renal proximal tubule epithelial cells

In Vitro Skeletal Muscle Model of PGM1 Deficiency Reveals Altered Energy Homeostasis.

Stress-dependent macromolecular crowding in the mitochondrial matrix.

The Hypoxia Response Pathway: A Potential Intervention Target in Parkinson's Disease?

Performance of TMRM and Mitotrackers in mitochondrial morphofunctional analysis of primary human skin fibroblasts.

SMDT1 variants impair EMRE-mediated mitochondrial calcium uptake in patients with muscle involvement.

Dimethyl itaconate induces long-term innate immune responses and confers protection against infection.

Ndufs4 knockout mouse models of Leigh syndrome: pathophysiology and intervention

The DEAD-box RNA helicase Dhx15 controls glycolysis and arbovirus replication in Aedes aegypti mosquito cells

Mechanisms and mathematical modeling of ROS production by the mitochondrial electron transport chain

The decylTPP mitochondria-targeting moiety lowers electron transport chain supercomplex levels in primary human skin fibroblasts.

Energy expenditure during cell spreading influences the cellular response to matrix stiffness

Mitochondria-targeted phenolic antioxidants induce ROS-protective pathways in primary human skin fibroblasts

Mitochondria-targeted phenolic antioxidants induce ROS-protective pathways in primary human skin fibroblasts

Collaborators (18)

Niamh Connolly

Tom Schirris

Gabor Juhasz

Richard Rodenburg

Merel Post

Han-Ming Shen

Scott Ayton

Frans Russel

Aneta Dobosz

Hans‐Uwe Simon

Alex Garanto

Pascal Miesen

Jon Lane

Peter H.G.M. Willems

Justyna Janikiewicz

Mariusz Wieckowski

Albert Quintana

Evert van Schothorst

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